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External Noise Effects in Silicon MOS Inversion Layer

Autor: Bernardo Spagnolo, Nicola Pizzolato, Stefano Spezia, Maria Antonietta Lodato, D. Persano Adorno

The effect of the addition of an external source of correlated noise on the electron transport in silicon MOS inversion layer, driven by a static electric field, has been investigated. The electron dynamics is simulated by a Monte Carlo procedure whi

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0036a4cad2615ad8163c1a5ec4ae2a0e
http://hdl.handle.net/10447/98879

ATM-deficiency-induced microglial activation promotes neurodegeneration in ataxia-telangiectasia.

Autor: Lai J; Division of Genetics and Genomics, Department of Pediatrics, Boston Children's Hospital, Boston, MA 02115, USA; The Manton Center for Orphan Disease Research, Boston Children's Hospital, Boston, MA 02115, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA; Program in Neuroscience, Harvard University, Boston, MA 02115, USA., Demirbas D; Division of Genetics and Genomics, Department of Pediatrics, Boston Children's Hospital, Boston, MA 02115, USA; The Manton Center for Orphan Disease Research, Boston Children's Hospital, Boston, MA 02115, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA., Kim J; Division of Genetics and Genomics, Department of Pediatrics, Boston Children's Hospital, Boston, MA 02115, USA; The Manton Center for Orphan Disease Research, Boston Children's Hospital, Boston, MA 02115, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA., Jeffries AM; Department of Molecular, Cell, and Cancer Biology, University of Massachusetts Medical School, Worcester, MA 01605, USA., Tolles A; Department of Molecular, Cell, and Cancer Biology, University of Massachusetts Medical School, Worcester, MA 01605, USA., Park J; Division of Genetics and Genomics, Department of Pediatrics, Boston Children's Hospital, Boston, MA 02115, USA; The Manton Center for Orphan Disease Research, Boston Children's Hospital, Boston, MA 02115, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA., Chittenden TW; Division of Genetics and Genomics, Department of Pediatrics, Boston Children's Hospital, Boston, MA 02115, USA; Computational Statistics and Bioinformatics Group, Genuity AI Research Institute, Genuity Science, Boston, MA 02114, USA., Buckley PG; Genuity Genomics Centre, Genuity Science, D18 K7W4 Dublin, Ireland., Yu TW; Division of Genetics and Genomics, Department of Pediatrics, Boston Children's Hospital, Boston, MA 02115, USA; The Manton Center for Orphan Disease Research, Boston Children's Hospital, Boston, MA 02115, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA., Lodato MA; Department of Molecular, Cell, and Cancer Biology, University of Massachusetts Medical School, Worcester, MA 01605, USA. Electronic address: michael.lodato@umassmed.edu., Lee EA; Division of Genetics and Genomics, Department of Pediatrics, Boston Children's Hospital, Boston, MA 02115, USA; The Manton Center for Orphan Disease Research, Boston Children's Hospital, Boston, MA 02115, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA. Electronic address: ealee@childrens.harvard.edu.

Publikováno v: Cell reports [Cell Rep] 2024 Jan 23; Vol. 43 (1), pp. 113622. Date of Electronic Publication: 2023 Dec 29.

Editorial: Somatic mutations, genome mosaicism and aging.

Autor: Lodato MA; Department of Molecular, Cell, and Cancer Biology, University of Massachusetts Chan Medical School, Worcester, MA, United States., Vijg J; Center for Single-Cell Omics, School of Public Health, Shanghai Jiao Tong University School of Medicine, Shanghai, China.; Department of Genetics, Albert Einstein College of Medicine, New York City, NY, United States.

Publikováno v: Frontiers in aging [Front Aging] 2023 Jan 09; Vol. 3, pp. 1115408. Date of Electronic Publication: 2023 Jan 09 (Print Publication: 2022).

Prevalence and mechanisms of somatic deletions in single human neurons during normal aging and in DNA repair disorders.

Autor: Kim J; Division of Genetics and Genomics, Boston Children's Hospital, Boston, MA, USA.; Manton Center for Orphan Disease, Boston Children's Hospital, Boston, MA, USA.; Department of Pediatrics, Harvard Medical School, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA.; Department of Biological Sciences, Sungkyunkwan University, Suwon, South Korea., Huang AY; Division of Genetics and Genomics, Boston Children's Hospital, Boston, MA, USA.; Manton Center for Orphan Disease, Boston Children's Hospital, Boston, MA, USA.; Department of Pediatrics, Harvard Medical School, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA., Johnson SL; Department of Molecular, Cell and Cancer Biology, University of Massachusetts Medical School, Worcester, MA, USA., Lai J; Division of Genetics and Genomics, Boston Children's Hospital, Boston, MA, USA.; Manton Center for Orphan Disease, Boston Children's Hospital, Boston, MA, USA.; Department of Pediatrics, Harvard Medical School, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA., Isacco L; Division of Genetics and Genomics, Boston Children's Hospital, Boston, MA, USA.; Manton Center for Orphan Disease, Boston Children's Hospital, Boston, MA, USA.; Department of Pediatrics, Harvard Medical School, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA.; Howard Hughes Medical Institute, Boston Children's Hospital, Boston, MA, USA.; Department of Neurology, Harvard Medical School, Boston, MA, USA., Jeffries AM; Department of Molecular, Cell and Cancer Biology, University of Massachusetts Medical School, Worcester, MA, USA., Miller MB; Division of Genetics and Genomics, Boston Children's Hospital, Boston, MA, USA.; Manton Center for Orphan Disease, Boston Children's Hospital, Boston, MA, USA.; Department of Pediatrics, Harvard Medical School, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA.; Howard Hughes Medical Institute, Boston Children's Hospital, Boston, MA, USA.; Department of Neurology, Harvard Medical School, Boston, MA, USA.; Department of Pathology, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA., Lodato MA; Division of Genetics and Genomics, Boston Children's Hospital, Boston, MA, USA.; Manton Center for Orphan Disease, Boston Children's Hospital, Boston, MA, USA.; Department of Pediatrics, Harvard Medical School, Boston, MA, USA.; Broad Institute of MIT and Harvard, Cambridge, MA, USA.; Department of Molecular, Cell and Cancer Biology, University of Massachusetts Medical School, Worcester, MA, USA.; Howard Hughes Medical Institute, Boston Children's Hospital, Boston, MA, USA.; Department of Neurology, Harvard Medical School, Boston, MA, USA., Walsh CA; Division of Genetics and Genomics, Boston Children's Hospital, Boston, MA, USA. christopher.walsh@childrens.harvard.edu.; Manton Center for Orphan Disease, Boston Children's Hospital, Boston, MA, USA. christopher.walsh@childrens.harvard.edu.; Department of Pediatrics, Harvard Medical School, Boston, MA, USA. christopher.walsh@childrens.harvard.edu.; Broad Institute of MIT and Harvard, Cambridge, MA, USA. christopher.walsh@childrens.harvard.edu.; Howard Hughes Medical Institute, Boston Children's Hospital, Boston, MA, USA. christopher.walsh@childrens.harvard.edu.; Department of Neurology, Harvard Medical School, Boston, MA, USA. christopher.walsh@childrens.harvard.edu., Lee EA; Division of Genetics and Genomics, Boston Children's Hospital, Boston, MA, USA. EAlice.Lee@childrens.harvard.edu.; Manton Center for Orphan Disease, Boston Children's Hospital, Boston, MA, USA. EAlice.Lee@childrens.harvard.edu.; Department of Pediatrics, Harvard Medical School, Boston, MA, USA. EAlice.Lee@childrens.harvard.edu.; Broad Institute of MIT and Harvard, Cambridge, MA, USA. EAlice.Lee@childrens.harvard.edu.

Publikováno v: Nature communications [Nat Commun] 2022 Oct 07; Vol. 13 (1), pp. 5918. Date of Electronic Publication: 2022 Oct 07.