Zobrazeno 1 - 10
of 210
pro vyhledávání: '"Lishmanov IuB"'
Publikováno v:
Europe PubMed Central
Experimental data indicate that moderate uncoupling oxidative phosphorylation induces reduction in production of reactive oxygen species (ROS) and promotes an increase in survival of neurons and cardiomyocytes under hypoxia and re-oxygenation conditi
Autor:
Ln, Maslov, Lishmanov IuB, Kolar F, Ag, Portnichenko, Podoksenov IuK, Igor Khaliulin, Wang H, Jm, Pei
Publikováno v:
Europe PubMed Central
The work covers the problem of hypoxic preconditioning (HP) carried out in isolated cardiomyocytes. Papers on delayed HP in vivo are comparatively few, and only some single works are devoted to early preconditioning in vivo. It has been established t
Publikováno v:
Bulletin of Experimental Biology and Medicine. 114:1596-1599
The system Fe(2+)-ascorbate was used for lipid peroxidation (LPO) activation in isolated heart tissue. The stimulation of free radical processes in the myocardium caused a rise in the level of LPO products and a significant lowering of that of prosta
Publikováno v:
Europe PubMed Central
Intraperitoneal administration of the sigma-receptor antagonist DuP 734 (1 mg/kg) 15 min before heart excision produces a decrease in the reperfusion heart contractility and prevented from the reperfusion induced cardiac cell lesion upon global ische
Publikováno v:
Bulletin of Experimental Biology and Medicine. 97:261-264
Intravenous injection of parathyroidine to intact rats in a dose of 2 Units/100 g bw provoked a hypotensive effect. The blockade of alpha-adrenoreceptors did not change the effect of parathyroid hormone. The stable analog of leu-enkephalin inhibited
Publikováno v:
Bulletin of Experimental Biology and Medicine. 99:535-537
It has been shown in experiments on albino rats that acute myocardial ischemia (AMI) produces a noticeable increase in excretion of adrenaline, noradrenaline, DOPA and dopamine with urine. Intraperitoneal injection of leu-enkephalin analogs (D-Ala2-L
Publikováno v:
Europe PubMed Central
It has been discussed literature data on the role for ORL1 (NOR) receptors in the regulation of function of gastrointestinal, respiratory, cardiovascular, immune, endocrine systems. In addition, it has been discussed a possibility of penetration of b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::ac0a0cc5e5a5287f8d6e8ae33a99e03c
http://europepmc.org/abstract/med/14743717
http://europepmc.org/abstract/med/14743717
Publikováno v:
Europe PubMed Central
As of now, the study of ventricular arrhythmias (VA) is of topical interest for modern arrhythmology and cardiology. These arrhythmias constitute about 30% of all the arrhythmias and have a diverse etiopathogenetic basis. Having no noticeable impact
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::7def34e4cac7aefce46f531a483b1909
http://europepmc.org/abstract/med/26165008
http://europepmc.org/abstract/med/26165008
Publikováno v:
Europe PubMed Central
Analysis of published data allows affirming that heat shock proteins (HSP) play an important role in the mechanism of cardioprotective effect of delayed preconditioning. However, HSP in all probability are non-end effectors but mediators of precondit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::981d3890862585aba01f0e332fefd7b8
http://europepmc.org/abstract/med/20583571
http://europepmc.org/abstract/med/20583571
Autor:
Ln, Maslov, Lishmanov IuB, Tv, Emel Ianova, Da, Prut, Kolar F, Ag, Portnichenko, Podoksenov IuK, Igor Khaliulin, Wang H, Jm, Pei
Publikováno v:
Europe PubMed Central
Analysis of published data indicates that delayed hypoxic preconditioning essentially increases a cardiac and brain tolerance to ischemia-reperfusion. There are no experimental data in the literature on the neuroprotective effect of early hypoxic pre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::bd99be69e4450badc7a4ae2fd595330e
http://europepmc.org/abstract/med/22027518
http://europepmc.org/abstract/med/22027518