Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Lisa Picard"'
Publikováno v:
The Journal of Trauma: Injury, Infection, and Critical Care. 38:175-184
The combination of burn and smoke inhalation was studied to determine if early hemodynamic and metabolic abnormalities would correspond with the degree of subsequent smoke-induced airways injury. Adult sheep (n = 45) given an 18% total body surface t
Publikováno v:
Surgery. 117:226-231
Background. Inflammation-induced disease as seen with trauma and infection can lead to increased lung oxidant activity resulting in cell membrane lipid peroxidation. Acute zymosan-induced peritonitis in rats produces lung inflammation, edema, and lip
Publikováno v:
Critical Care Medicine. 23:171-178
Objective To study the early (first 24 hrs) effect of increasing lung exposure to smoke on the hemodynamic response to a modest body burn. Design A prospective randomized study. Setting Laboratory at a university medical center. Subjects Thirty-two a
Publikováno v:
Inflammation. 18:499-510
Acute zymosan-induced peritonitis in rats produces lung inflammation and lipid peroxidation. The effect of this process on plasma and lung tissue ascorbic acid was determined, as was the effect of infusing 150 mg/kg of ascorbic acid immediately after
Publikováno v:
Shock. 1:101-107
We determined the oxidant activity in lung airways, parenchyma, and systemic tissues in response to smoke inhalation, comparing lipid peroxidation with physiologic and histologic change. Adult sheep were given a controlled amount of cooled smoke from
Publikováno v:
Shock (Augusta, Ga.). 1(4)
We determined whether plasma levels of reduced glutathione (GSH) and oxidized glutathione (GSSG) accurately reflect the tissue GSH and GSSG levels in lung and liver during a progressive acute inflammation-induced increased oxidant activity. We also d
Publikováno v:
Critical care medicine. 21(12)
OBJECTIVE: We compared the effect of a modest smoke inhalation injury, a burn injury alone, and a smoke inhalation injury plus a body burn, on the degree of lung oxidant-induced lipid peroxidation and lung injury. DESIGN: Prospective animal study wit