Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Lisa M. Morey"'
Publikováno v:
Toxicology Reports, Vol 2, Iss C, Pp 817-823 (2015)
Exposure to estrogenic compounds has been shown to epigenetically reprogram the prostate and may contribute to prostate cancer. The goal of this study was to determine the effect of physiological doses of estradiol and bisphenol A (BPA) on the expres
Externí odkaz:
https://doaj.org/article/dc1c665eb8cb4d3e9c59ead2b32a0a5d
Publikováno v:
Dose-Response, Vol 14 (2016)
Exposure to estrogenic compounds has been shown to epigenetically reprogram the female reproductive tract and may contribute to ovarian cancer. The goal of this study was to compare the effect of estradiol or bisphenol A (BPA) on the expression of hi
Externí odkaz:
https://doaj.org/article/f47036a11ee0478da7db749f359dcf67
Autor:
Karen E. Knudsen, Jason Barrera, Lisa M. Morey, Janet K. Hess-Wilson, Khanh H. Cao, Erik S. Knudsen, Clay E.S. Comstock, Ankur Sharma
Supplementary Figures 1-3 from Retinoblastoma Tumor Suppressor Status Is a Critical Determinant of Therapeutic Response in Prostate Cancer Cells
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::157da18b7817176b687595591d68753b
https://doi.org/10.1158/0008-5472.22367948.v1
https://doi.org/10.1158/0008-5472.22367948.v1
Autor:
Karen E. Knudsen, Jason Barrera, Lisa M. Morey, Janet K. Hess-Wilson, Khanh H. Cao, Erik S. Knudsen, Clay E.S. Comstock, Ankur Sharma
The retinoblastoma tumor suppressor protein (RB), a critical mediator of cell cycle progression, is functionally inactivated in the majority of human cancers, including prostatic adenocarcinoma. The importance of RB tumor suppressor function in this
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2da0c9bb0467c3544b64ceaacdf6615c
https://doi.org/10.1158/0008-5472.c.6495749.v1
https://doi.org/10.1158/0008-5472.c.6495749.v1
Autor:
Karen E. Knudsen, Jason Barrera, Lisa M. Morey, Janet K. Hess-Wilson, Khanh H. Cao, Erik S. Knudsen, Clay E.S. Comstock, Ankur Sharma
Supplementary Legends 1-3 from Retinoblastoma Tumor Suppressor Status Is a Critical Determinant of Therapeutic Response in Prostate Cancer Cells
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5f2afac69fe1ba9c73433a456a9ac0cd
https://doi.org/10.1158/0008-5472.22367945
https://doi.org/10.1158/0008-5472.22367945
Publikováno v:
Toxicology Reports, Vol 2, Iss C, Pp 817-823 (2015)
Exposure to estrogenic compounds has been shown to epigenetically reprogram the prostate and may contribute to prostate cancer. The goal of this study was to determine the effect of physiological doses of estradiol and bisphenol A (BPA) on the expres
Publikováno v:
Dose-Response, Vol 14 (2016)
Dose-Response
Dose-Response
Exposure to estrogenic compounds has been shown to epigenetically reprogram the female reproductive tract and may contribute to ovarian cancer. The goal of this study was to compare the effect of estradiol or bisphenol A (BPA) on the expression of hi
Publikováno v:
Endocrinology. 153:42-55
Evidence supporting an early origin of prostate cancer is growing. We demonstrated previously that brief exposure of neonatal rats to estradiol or bisphenol A elevated their risk of developing precancerous lesions in the prostate upon androgen-suppor
Autor:
Diane E. Merry, Yuhong Liu, Matthew J. Schiewer, Craig J. Burd, Lisa M. Morey, Shuk-Mei Ho, Karen E. Knudsen
Publikováno v:
Oncogene
Regulation of the androgen receptor (AR) is critical to prostate cancer (PCa) development; therefore, AR is the first line therapeutic target for disseminated tumors. Cell cycle dependent accumulation of cyclin D1 negatively modulates the transcripti
Autor:
Clay E.S. Comstock, E M Groh, Yuan-Liang Wang, Lisa M. Morey, Karen E. Knudsen, N A Olshavsky, Monica P. Revelo, Jaroslaw Meller, Craig J. Burd
Publikováno v:
Oncogene. 27:3111-3121
Prostate cancer (PCa) cell proliferation is dependent on activation of the androgen receptor (AR), a ligand-dependent transcription factor. AR activation controls G1-S phase progression through fostering enhanced translation of the D-type cyclins, wh