Zobrazeno 1 - 10
of 32
pro vyhledávání: '"Lisa J. Glickstein"'
Publikováno v:
Arthritis & Rheumatism. 64:1497-1507
Objective Single-nucleotide polymorphisms (SNPs) that alter immune function, inflammatory responses, and disease susceptibility have been identified in several genes encoding Toll-like receptors (TLRs). The TLR SNPs with the best evidence of an effec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bb101eef931a4959f3038da8a0848721
https://doi.org/10.1002/art.34383
https://doi.org/10.1002/art.34383
Publikováno v:
Molecular Immunology. 45:2323-2332
Antibiotic-refractory Lyme arthritis is believed to result from an infection-induced autoimmune response triggered by the spirochete Borrelia burgdorferi ( Bb ). Disease susceptibility is associated with the HLA alleles DRB1*0101, 0401, 0402, 0404, 0
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ad9a2b1af7cc44bdf74b21edc3811ca2
https://doi.org/10.1016/j.molimm.2007.11.010
https://doi.org/10.1016/j.molimm.2007.11.010
Autor:
Allen C. Steere, Rendi Murphree Bacon, Priya Kannian, Barbara J. B. Johnson, Lisa J. Glickstein, Gail McHugh
Publikováno v:
Arthritis & Rheumatism. 56:4216-4225
Objective To compare the pattern of antibody responses to Borrelia burgdorferi in patients with antibiotic-refractory, antibiotic-responsive, or non–antibiotic-treated Lyme arthritis as an indirect measure of spirochetal persistence or eradication.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::60ffc7e75b8de4711b760548bebb41e2
https://doi.org/10.1002/art.23135
https://doi.org/10.1002/art.23135
Autor:
Lisa J. Glickstein, Priya Kannian, Allen C. Steere, Gerald T. Nepom, William W. Kwok, Elise E. Drouin
Publikováno v:
The Journal of Immunology. 179:6336-6342
Synovitis in patients with antibiotic-refractory Lyme arthritis persists for months to several years after antibiotic therapy. This course, which may result from infection-induced autoimmunity, is associated with T cell recognition of Borrelia burgdo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a3eaa6f3e59e4ade9bd7dbbce5a60468
https://doi.org/10.4049/jimmunol.179.9.6336
https://doi.org/10.4049/jimmunol.179.9.6336
Publikováno v:
Arthritis & Rheumatism. 56:1325-1335
Objective To investigate the possible role of chemokines and cytokines in the pathogenesis of Lyme arthritis. Methods Using cytometric bead array and flow cytometry techniques, chemokine and cytokine levels were determined in 65 synovial fluid (SF) s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6d8e67032cab0765156617c540664ca7
https://doi.org/10.1002/art.22441
https://doi.org/10.1002/art.22441
Autor:
Gail McHugh, Lisa J. Glickstein, Vijay K. Sikand, Kathryn L. Jones, Allen C. Steere, Nitin Damle
Publikováno v:
Journal of Clinical Microbiology. 44:4407-4413
Three genetic markers of Borrelia burgdorferi have been associated with disseminated disease: the OspC type, the 16S-23S rRNA intergenic spacer type (RST), and vlsE . Here, we modified previous methods so as to identify the three markers by PCR and r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e38655d8de48267af6aadc73438cb3c3
https://doi.org/10.1128/jcm.01077-06
https://doi.org/10.1128/jcm.01077-06
Autor:
Lisa J. Glickstein, Jenifer Coburn
Publikováno v:
The American Journal of Tropical Medicine and Hygiene. 75:964-967
Susceptibility to Borrelia burgdorferi infection and subsequent arthritis is genetically determined in mice and determined by innate immunity. Accordingly, macrophage responses to B. burgdorferi challenge may differ between mouse strains. Bone marrow
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ef6e4f57a7d9446091db4ac08a54ce87
https://doi.org/10.4269/ajtmh.2006.75.964
https://doi.org/10.4269/ajtmh.2006.75.964
Publikováno v:
Journal of Autoimmunity. 23:281-292
Treatment-resistant Lyme arthritis, which may result from infection-induced autoimmunity, is associated with reactivity to a T cell epitope of outer-surface protein A (OspA 161–175 ) of Borrelia burgdorferi sensu stricto ( Bb ). This syndrome has b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::14b4e926a813520a912a6fca2633879e
https://doi.org/10.1016/j.jaut.2004.06.005
https://doi.org/10.1016/j.jaut.2004.06.005
Publikováno v:
Journal of Clinical Investigation. 113:1093-1101
Since its identification nearly 30 years ago, Lyme disease has continued to spread, and there have been increasing numbers of cases in the northeastern and north central US. The Lyme disease agent, Borrelia burgdorferi, causes infection by migration
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dafdaf4ce3c5f8916425690304eeb73f
https://doi.org/10.1172/jci21681
https://doi.org/10.1172/jci21681
Publikováno v:
Infection and Immunity. 69:3737-3743
Lyme arthritis is the most common complication following infection of human individuals withBorrelia burgdorferisensu stricto. In mice,B. burgdorferiinfection leads to arthritis of the tibiotarsal joints. Arthritis severity in mice is under host gene
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::393058eaa0e52dc1d71084cb6013ab83
https://doi.org/10.1128/iai.69.6.3737-3743.2001
https://doi.org/10.1128/iai.69.6.3737-3743.2001