Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Lisa B. Carter"'
Publikováno v:
Neurobiology of Disease, Vol 21, Iss 3, Pp 522-530 (2006)
General anesthetics cause widespread apoptotic neurodegeneration in many regions of the developing rat brain. The activation of mitochondria-dependent apoptotic pathway is important in the early stages of anesthesia-induced developmental neuroapoptos
Externí odkaz:
https://doaj.org/article/f828c28210f54e119eb54c66e4d2acd8
Publikováno v:
Brain Pathology. 18:198-210
Exposure of the immature brain to general anesthesia is common. The safety of this practice has recently been challenged in view of evidence that general anesthetics can damage developing mammalian neurons. Initial reports on immature rats raised cri
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::26f5e8847a43a255ee734e9a290bb771
https://doi.org/10.1111/j.1750-3639.2007.00116.x
https://doi.org/10.1111/j.1750-3639.2007.00116.x
Publikováno v:
Annals of the New York Academy of Sciences. 1122:69-82
Frequent exposure of children to general anesthesia is common practice in modern medicine. Although previously unrecognized, recent in vitro and in vivo animal studies suggest that exposure to clinically relevant general anesthetics at the peak of br
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5dea6046eeff3ff2df183cae5180b97e
https://doi.org/10.1196/annals.1403.005
https://doi.org/10.1196/annals.1403.005
Publikováno v:
Apoptosis. 11:1603-1615
Brain-derived neurotrophic factor (BDNF) is important in supporting neuronal development. BDNF imbalance due to excessive neuronal inhibition can result in the apoptotic degeneration of developing neurons. Since general anesthetics cause profound dep
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a200de279acdf21ed592ac3096c10923
https://doi.org/10.1007/s10495-006-8762-3
https://doi.org/10.1007/s10495-006-8762-3
Publikováno v:
Neuroscience. 135:815-827
It was shown recently that exposure of the developing rat brain during the peak of synaptogenesis to commonly used general anesthetics can trigger widespread apoptotic neurodegeneration in many regions of the developing rat brain and persistent learn
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8a1fb512748283bf30547946f42b2628
https://doi.org/10.1016/j.neuroscience.2005.03.064
https://doi.org/10.1016/j.neuroscience.2005.03.064
Exposure of the immature brain to general anesthesia is common. The safety of this practice has recently been challenged in view of evidence that general anesthetics can damage developing mammalian neurons. Initial reports on immature rats raised cri
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::617f54a664d847eac2cdc2433e314d7c
http://hdl.handle.net/11577/2491506
http://hdl.handle.net/11577/2491506
Publikováno v:
Neurobiology of Disease, Vol 21, Iss 3, Pp 522-530 (2006)
General anesthetics cause widespread apoptotic neurodegeneration in many regions of the developing rat brain. The activation of mitochondria-dependent apoptotic pathway is important in the early stages of anesthesia-induced developmental neuroapoptos
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::deceab3ea6064b38c72b28abdc9aa796
https://pubmed.ncbi.nlm.nih.gov/16289675
https://pubmed.ncbi.nlm.nih.gov/16289675
Publikováno v:
Neurobiology of aging. 26(6)
Nitrous oxide (N 2 O) and ketamine are common general anesthetics and antagonists of N -methyl- d -aspartate (NMDA) glutamate receptors. In clinically relevant concentrations, they induce a psychotomimetic reaction in humans and pathomorphological ch
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::54f5622be02f27be74e693e63b257a41
https://pubmed.ncbi.nlm.nih.gov/15718054
https://pubmed.ncbi.nlm.nih.gov/15718054
Publikováno v:
Annals of the New York Academy of Sciences. 993
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::25243f16068fc453d38780460d924a87
https://pubmed.ncbi.nlm.nih.gov/12853302
https://pubmed.ncbi.nlm.nih.gov/12853302