Zobrazeno 1 - 10
of 47
pro vyhledávání: '"Lisa A. Elferink"'
Autor:
Jeannine Garnett, Vaibhav Chumbalkar, Brian Vaillant, Anupama E. Gururaj, Kristen S. Hill, Khatri Latha, Jun Yao, Waldemar Priebe, Howard Colman, Lisa A. Elferink, Oliver Bogler
Publikováno v:
Neoplasia: An International Journal for Oncology Research, Vol 15, Iss 1, Pp 73-84 (2013)
The hepatocyte growth factor receptor (c-Met) and a constitutively active mutant of the epidermal growth factor receptor (ΔEGFR/EGFRvIII) are frequently overexpressed in glioblastoma (GBM) and promote tumorigenesis. The mechanisms underlying elevate
Externí odkaz:
https://doaj.org/article/6aac99f91bf44a98a0902ce4778a0265
Autor:
Ivana Gaziova, Daniel Jackson, Paul J Boor, Dwayne Carter, Zobeida Cruz-Monserrate, Cornelis J Elferink, Aditya D Joshi, Bhupendra Kaphalia, Craig D Logsdon, Karen Pereira de Castro, Lynn Soong, Xinrong Tao, Suimin Qiu, Lisa A Elferink
Publikováno v:
PLoS ONE, Vol 11, Iss 10, p e0165485 (2016)
Acinar cells represent the primary target in necroinflammatory diseases of the pancreas, including pancreatitis. The signaling pathways guiding acinar cell repair and regeneration following injury remain poorly understood. The purpose of this study w
Externí odkaz:
https://doaj.org/article/418a58ff5b974aa3a5e84a139e093f08
Autor:
Noelle C. Anastasio, John A. Allen, Holly L. Chapman, Kathryn A. Cunningham, Lisa A. Elferink, Daniel E. Felsing, Brionna D. Davis-Reyes, F. Gerard Moeller, Michelle A. Land
Publikováno v:
Scientific Reports, Vol 9, Iss 1, Pp 1-15 (2019)
A non-synonymous single nucleotide polymorphism of the human serotonin 5-HT2C receptor (5-HT2CR) gene that converts a cysteine to a serine at amino acid codon 23 (Cys23Ser) appears to impact 5-HT2CR pharmacology at a cellular and systems level. We hy
Autor:
Michelle A, Land, Holly L, Chapman, Brionna D, Davis-Reyes, Daniel E, Felsing, John A, Allen, F Gerard, Moeller, Lisa A, Elferink, Kathryn A, Cunningham, Noelle C, Anastasio
Publikováno v:
Scientific Reports
A non-synonymous single nucleotide polymorphism of the human serotonin 5-HT2C receptor (5-HT2CR) gene that converts a cysteine to a serine at amino acid codon 23 (Cys23Ser) appears to impact 5-HT2CR pharmacology at a cellular and systems level. We hy
Autor:
Cornelis J. Elferink, Zobeida Cruz-Monserrate, Bhupendra S. Kaphalia, Karen Pereira de Castro, Lisa A. Elferink, Lynn Soong, Aditya D. Joshi, Dwayne Carter, Paul J. Boor, Daniel Jackson, Craig D. Logsdon, Xinrong Tao, Ivana Gaziova, Suimin Qiu
Publikováno v:
PLoS ONE
PLoS ONE, Vol 11, Iss 10, p e0165485 (2016)
PLoS ONE, Vol 11, Iss 10, p e0165485 (2016)
Acinar cells represent the primary target in necroinflammatory diseases of the pancreas, including pancreatitis. The signaling pathways guiding acinar cell repair and regeneration following injury remain poorly understood. The purpose of this study w
Autor:
Lisa A. Elferink, Vicente A. Resto
Publikováno v:
Journal of Signal Transduction
Molecular therapeutics for treating epidermal growth factor receptor-(EGFR-) expressing cancers are a specific method for treating cancers compared to general cell loss with standard cytotoxic therapeutics. However, the finding that resistance to suc
Publikováno v:
Journal of Neurochemistry. 72:1821-1831
Synaptotagmin IV (Syt IV) is an immediate early gene induced by depolarization in rat PC12 cells and in rat hippocampus. We prepared an antiserum to Syt IV protein. The 46-kDa Syt IV protein is nearly undetectable by western blotting in unstimulated
Publikováno v:
Journal of Biological Chemistry. 282:16764-16775
Lysosomal degradation of the receptor-tyrosine kinase cMet requires receptor ubiquitination by the E3 ubiquitin ligase Cbl followed by clathrin-dependent internalization. A role for Cbl as an adaptor for cMet internalization has been previously repor
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 1270
The tyrosine kinase MET, a receptor for hepatocyte growth factor, is a key regulator for normal development and organ renewal via stem cell maintenance. Dysregulated MET signaling contributes to tumor progression and metastasis and is considered a po
Publikováno v:
Membrane Trafficking ISBN: 9781493923083
The tyrosine kinase MET, a receptor for hepatocyte growth factor, is a key regulator for normal development and organ renewal via stem cell maintenance. Dysregulated MET signaling contributes to tumor progression and metastasis and is considered a po
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b09540187c006ec240dbc119fc9f98a9
https://doi.org/10.1007/978-1-4939-2309-0_26
https://doi.org/10.1007/978-1-4939-2309-0_26