Zobrazeno 1 - 10 of 21 pro vyhledávání: '"Linguo Pei"'
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Akademický článek
Corticosterone rather than ethanol epigenetic programmed testicular dysplasia caused by prenatal ethanol exposure in male offspring rats
Autor: Min Liu, Qi Zhang, Linguo Pei, Yunfei Zou, Guanghui Chen, Hui Wang
Publikováno v: Epigenetics, Vol 14, Iss 3, Pp 245-259 (2019)
Prenatal ethanol exposure (PEE) could affect offspring’s testicular development. This study aimed to illuminate its intrauterine origin and the programming mechanism caused by PEE. Pregnant Wistar rats were given ethanol (4 g/kg.d) by gavage admini
Externí odkaz: https://doaj.org/article/02163f6f14bd4471890ec15b1ffeef38
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2
Activation of α-7 Nicotinic Acetylcholine Receptor Attenuates Cardiac Inflammation Through NLRP3/Caspase-1/IL-18 Pathway
Autor: Zijing Guo, Bin Tan, Junjie Wang, Weijun Tang, Linguo Pei, Yisi Chen, Jun Zhang
Publikováno v: Biochemical Genetics. 60:1333-1345
Activation of α-7 nicotinic acetylcholine receptor (α7nAChR) receptor might induce cardiac inflammation, cardiac remodeling, and dysfunction. In this regard, this study aims to clarify the role and mechanism of α7nAChR in the process of cardiac in
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::976f450101510c57ff115e55500fc207
https://doi.org/10.1007/s10528-021-10162-8
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3
Low miR-92a-3p in oocytes mediates the multigenerational and transgenerational inheritance of poor cartilage quality in rat induced by prenatal dexamethasone exposure
Autor: Kai Tie, Zhe Zhao, Zhixin Wu, Jun Qin, Jinzhi Zhang, Linguo Pei, Hui Wang, Liaobin Chen
Publikováno v: Biochemical pharmacology. 203
An adverse environment during pregnancy leads to intrauterine programming changes in multiple generations, resulting in the multigenerational inheritance of abnormal phenotype. Here, we reported the multigenerational inheritance of poor articular car
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::46ab61c510ba82b9926b305875cdce7a
https://pubmed.ncbi.nlm.nih.gov/35917869
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4
Prenatal ethanol exposure induced disorder of hypothalamic-pituitary-adrenal axis-associated neuroendocrine metabolic programming alteration and dysfunction of glucose and lipid metabolism in 40-week-old female offspring rats
Autor: Liping Xia, Linguo Pei, Hui Wang, Chao Yuan, Zhexiao Jiao, Yanjuan Zhao, Yu Guo
Publikováno v: Reproductive Toxicology. 94:48-54
This study was designed to demonstrate disorder of hypothalamic-pituitary-adrenal (HPA) axis-associated neuroendocrine metabolic programming alteration and dysfunction of glucose and lipid metabolism induced by prenatal ethanol exposure (PEE) in post
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3001db7ebb538e1d1197ca463f946600
https://doi.org/10.1016/j.reprotox.2020.04.075
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5
Corticosterone rather than ethanol epigenetic programmed testicular dysplasia caused by prenatal ethanol exposure in male offspring rats
Autor: Yun-Fei Zou, Linguo Pei, Hui Wang, Min Liu, Guanghui Chen, Qi Zhang
Publikováno v: Epigenetics. 14:245-259
Prenatal ethanol exposure (PEE) could affect offspring’s testicular development. This study aimed to illuminate its intrauterine origin and the programming mechanism caused by PEE. Pregnant Wistar rats were given ethanol (4 g/kg.d) by gavage admini
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3dec92f8f1d18ba3dd041df470760c02
https://doi.org/10.1080/15592294.2019.1581595
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6
Excitotoxicity and compensatory upregulation of GAD67 in fetal rat hippocampus caused by prenatal nicotine exposure are associated with inhibition of the BDNF pathway
Autor: Hui Wang, Linguo Pei, Zhexiao Jiao, Dan Xu, Yun Pei, Wanting Dong, Xia He
Publikováno v: Food and Chemical Toxicology. 123:314-325
Prenatal nicotine exposure (PNE) can cause hypersensitivity of hypothalamic-pituitary-adrenal (HPA) axis in offspring with intrauterine growth retardation. The purpose of this study was to explore the original mechanism of intrauterine development th
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be8a1c4bd3bd1a915cddf26517984fcf
https://doi.org/10.1016/j.fct.2018.10.062
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7
Articular damages in multi-generational female offspring due to prenatal caffeine exposure correlates with H3K9 deacetylation of TGFβ signaling pathway
Autor: Hui Wang, Zhe Zhao, Liaobin Chen, Linguo Pei, Jacques Magdalou, Hanwen Luo, Zheng He, Jun Qin
Publikováno v: Toxicology
Toxicology, Elsevier, 2020, 442, pp.152533. ⟨10.1016/j.tox.2020.152533⟩
Adverse environment during pregnancy could lead to maternal glucocorticoid overexposure in utero, and then induce the intrauterine growth retardation (IUGR) and the programmed change in cartilage development. The transforming growth factor β (TGFβ)
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::71852a9798418e4789a45d5f9b72cb2c
https://hal.univ-lorraine.fr/hal-02969249
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10
cAMP/PKA/EGR1 signaling mediates the molecular mechanism of ethanol-induced inhibition of placental 11β-HSD2 expression
Autor: Yuanzhen Zhang, Feng Lv, Wei Zhang, Wen Huang, Hui Wang, Linguo Pei, Luting Yu, Jin Zhou, Guohui Zhang
Publikováno v: Toxicology and Applied Pharmacology. 352:77-86
It is known that inhibiting 11β-hydroxysteroid dehydrogenase type 2 (11β-HSD2) expression in the placenta can cause fetal over-exposure to maternal glucocorticoids and induce intrauterine growth restriction (IUGR); these effects ultimately increase
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b9742cc6293b5c28afb9cb6648474cbc
https://doi.org/10.1016/j.taap.2018.05.029
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