Zobrazeno 1 - 10
of 17
pro vyhledávání: '"LingTao Luo"'
Autor:
Junjia Liu, Xinyi Lu, Siyu Zeng, Rong Fu, Xindong Wang, Lingtao Luo, Ting Huang, Xusheng Deng, Hualei Zheng, Shaoqian Ma, Dan Ning, Lili Zong, Shu-Hai Lin, Yongyou Zhang
Publikováno v:
Redox Biology, Vol 71, Iss , Pp 103118- (2024)
The induction of ferroptosis is promising for cancer therapy. However, the mechanisms enabling cancer cells to evade ferroptosis, particularly in low-cystine environments, remain elusive. Our study delves into the intricate regulatory mechanisms of A
Externí odkaz:
https://doaj.org/article/838f9b3cff53424b8a47bc84edd51b3f
Autor:
HaiFeng Jiang, WeiChao Su, HaiXing Wang, ChunYing Luo, YaTao Wang, LinJun Zhang, LingTao Luo, ZeBin Lu, DongYan Shen, GuoQiang Su
Publikováno v:
Heliyon, Vol 10, Iss 3, Pp e24807- (2024)
Colorectal Carcinoma (CRC) is one of the most common malignant tumors of the digestive tract, with a high mortality rate. DPY30 is one of the core subunits of the histone methyltransferase complex, which was involved in many cancer processes. However
Externí odkaz:
https://doaj.org/article/6f44af60ad41465db582a72e62818463
Autor:
Yongzhi Wang, Lingtao Luo, Oscar Ö. Braun, Eva Norström, Su Zhang, Matthias Mörgelin, Henrik Thorlacius
Publikováno v:
Journal of Cellular Physiology. 233:1051-1060
Sepsis is associated with dysfunctional coagulation. Recent data suggest that platelets play a role in sepsis by promoting neutrophil accumulation. Herein, we show that cecal ligation and puncture (CLP) triggered systemic inflammation, which is chara
Autor:
Heiko Herwald, Oscar Ö. Braun, Lingtao Luo, Raed Madhi, Johannes Westman, Henrik Thorlacius, Matthias Mörgelin, Yongzhi Wang
Publikováno v:
Scientific Reports
Scientific Reports, Vol 8, Iss 1, Pp 1-14 (2018)
Scientific Reports, Vol 8, Iss 1, Pp 1-14 (2018)
Abdominal sepsis is associated with dysfunctional hemostasis. Thrombin generation (TG) is a rate-limiting step in systemic coagulation. Neutrophils can expell neutrophil extracellular traps (NETs) and/or microparticles (MPs) although their role in pa
Autor:
Yongzhi Wang, Lingtao Luo, Johanna Puegge, Jonas Roller, Su Zhang, Henrik Thorlacius, Brigitte Vollmar
Publikováno v:
European Surgical Research. 56:19-31
Background: Extracellular histones released during cell damage have the capacity to cause tissue injury associated with increased leukocyte accumulation. However, the molecular mechanisms regulating histone-induced leukocyte recruitment remain elusiv
Publikováno v:
Journal of Cellular Physiology. 231:370-376
Accumulating data suggest that platelets not only regulate thrombosis and haemostasis but also inflammatory processes. Platelets contain numerous potent pro-inflammatory compounds, including the chemokines CCL5 and CXCL4 although their role in acute
Autor:
Ingvar Syk, Enming Zhang, Henrik Thorlacius, Lingtao Luo, Yongzhi Wang, Su Zhang, Milladur Rahman
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 307:L586-L596
Excessive neutrophil activation is a major component in septic lung injury. Neutrophil-derived DNA may form extracellular traps in response to bacterial invasions. The aim of the present study was to investigate the potential role of neutrophil extra
Publikováno v:
Infection and Immunity. 82:3275-3288
The signaling mechanisms regulating neutrophil recruitment, systemic inflammation, and T-cell dysfunction in polymicrobial sepsis are not clear. This study explored the potential involvement of the calcium/calcineurin-dependent transcription factor,
Publikováno v:
European journal of pharmacology. 746
Neutrophil recruitment is known to be a rate-limiting step in mediating tissue injury in severe acute pancreatitis (AP). However, the signalling mechanisms controlling inflammation and organ damage in AP remain elusive. Herein, we examined the role o
Publikováno v:
European journal of pharmacology. 741
Excessive neutrophil recruitment in the colon is a major feature in acute colitis although the signaling mechanisms behind colonic recruitment of neutrophils remain elusive. Herein, we hypothesized that Rac1 activity might play an important role in n