Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Ling-Xun Duan"'
Autor:
Jennifer M. P. Woo, Norio Horiike, M. A. Feitelson, B. Sun, K. Steensma, Ling-Xun Duan, J. Guo, Baruch S. Blumberg
Publikováno v:
Journal of Infectious Diseases. 172:713-722
The etiology of non-A, non-B hepatitis (NANBH) in renal dialysis patients was determined. Hepatitis C virus was present in many, but its appearance did not correlate with elevated alanine aminotransaminase. When sera from these patients were tested f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d8656a7f6665dda08a95ceb9eb1d1e98
https://doi.org/10.1093/infdis/172.3.713
https://doi.org/10.1093/infdis/172.3.713
Publikováno v:
Gastroenterology. 108:1810-1819
Background/Aims: The finding of antibodies against the polymerase of hepatitis B virus in renal dialysis patients before the incubation phase of infection implies underlying virus replication. Hence, the aim of the study was to test for virus during
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::392c6a6bca146b8e97c2031249617919
https://doi.org/10.1016/0016-5085(95)90144-2
https://doi.org/10.1016/0016-5085(95)90144-2
Autor:
Ling-Xun Duan, Norio Horiike, William F. Carman, Baruch S. Blumberg, M. A. Feitelson, Howard C. Thomas, J. Guo, G. Mcintyre
Publikováno v:
Journal of Viral Hepatitis. 2:19-31
SUMMARY. Hepatitis B virus (HBV) variants containing mutations within the X and the precore regions of the viral genome were demonstrated by polymerase chain reaction (PCR) amplification and DNA sequencing in renal dialysis patients with different se
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e86c8341db83b89a3ff281684062b09b
https://doi.org/10.1111/j.1365-2893.1995.tb00068.x
https://doi.org/10.1111/j.1365-2893.1995.tb00068.x
Publikováno v:
International Journal of Cancer. 55:571-576
Chronic infection with hepatitis-B virus (HBV) is associated with high risk for the development of hepatocellular carcinoma (HCC). Several studies have implicated that the X gene product(s) of HBV are important to the pathogenesis of HCC. This study
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4a6342a6ebce402d9ed11c988a1d447a
https://doi.org/10.1002/ijc.2910550409
https://doi.org/10.1002/ijc.2910550409
Publikováno v:
Journal of Hepatology. 17:S24-S34
This study tests the hypothesis that woodchuck hepatitis virus encoded X-antigen expression correlates with viral replication, with hepatitis, or with both. Paired liver and serum samples from each of 55 infected woodchucks were used. Seven of 8 carr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0444c5cb620c342c2a3e62f701e2b32a
https://doi.org/10.1016/s0168-8278(05)80420-9
https://doi.org/10.1016/s0168-8278(05)80420-9
Publikováno v:
Virology. 180:430-433
The woodchuck hepatitis virus (WHV) polymerase (pol)-encoded polypeptide(s), obtained from purified virus nucleo-capsid particles, have been characterized by Western blotting. Peptide antibodies to amino-terminal (residues 32–45, WHV pol-6) and car
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4769614b09ebff33c35bd5c6eb82f0e3
https://doi.org/10.1016/0042-6822(91)90052-d
https://doi.org/10.1016/0042-6822(91)90052-d
Publikováno v:
The Journal of general virology. 82(Pt 1)
The hepatitis B virus-encoded X antigen (HBxAg) may contribute to the development of liver cancer, in part, by stimulating the growth and survival of infected cells in the face of ongoing immune responses. Given that the Fas ligand/receptor system co
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4f7ab1540eea2089413fb637160351f3
https://pubmed.ncbi.nlm.nih.gov/11125170
https://pubmed.ncbi.nlm.nih.gov/11125170
Publikováno v:
Journal of hepatology. 13
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f104e81eed6eb9277d803cfe451aa418
https://pubmed.ncbi.nlm.nih.gov/1822515
https://pubmed.ncbi.nlm.nih.gov/1822515