Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Linda Molla"'
Autor:
Gerardo A Morfini, Daryl A Bosco, Hannah Brown, Rodolfo Gatto, Agnieszka Kaminska, Yuyu Song, Linda Molla, Lisa Baker, M Natalia Marangoni, Sarah Berth, Ehsan Tavassoli, Carolina Bagnato, Ashutosh Tiwari, Lawrence J Hayward, Gustavo F Pigino, D Martin Watterson, Chun-Fang Huang, Gary Banker, Robert H Brown, Scott T Brady
Publikováno v:
PLoS ONE, Vol 8, Iss 6, p e65235 (2013)
Dying-back degeneration of motor neuron axons represents an established feature of familial amyotrophic lateral sclerosis (FALS) associated with superoxide dismutase 1 (SOD1) mutations, but axon-autonomous effects of pathogenic SOD1 remained undefine
Externí odkaz:
https://doaj.org/article/c19ff8f0c04447fd912d1b2f9f9ca560
Autor:
Linda Molla, Ignacio L. Ibarra, Javier M. Di Noia, F. Nina Papavasiliou, Jana Ridani, Sandra Ruf, Judith B. Zaugg, Alin Vonica, Poorani Ganesh Subramani, Jonathan Boulais, Dewi Harjanto, Christoph Dieterich, Jose Paulo Lorenzo
The activation induced cytidine deaminase/apolipoprotein B editing complex (AID/APOBEC) family comprises several nucleic acid editors with roles ranging from antibody diversification to mRNA editing. APOBEC2, an evolutionarily conserved member of thi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::eae82cd2a07d02321309a563bddb7ea0
https://doi.org/10.1101/2020.07.29.223594
https://doi.org/10.1101/2020.07.29.223594
Autor:
M. Natalia Marangoni, Agnieszka Kaminska, Yuyu Song, Scott T. Brady, Ehsan Tavassoli, Sarah H. Berth, Rodolfo G. Gatto, Daryl A. Bosco, Ashutosh Tiwari, Robert H. Brown, Chun Fang Huang, Carolina Bagnato, Lisa Baker, D. Martin Watterson, Gerardo Morfini, Hannah E. Brown, Gustavo Pigino, Gary Banker, Linda Molla, Lawrence J. Hayward
Publikováno v:
PLoS ONE
PLoS ONE, Vol 8, Iss 6, p e65235 (2013)
PLoS ONE, Vol 8, Iss 6, p e65235 (2013)
Dying-back degeneration of motor neuron axons represents an established feature of familial amyotrophic lateral sclerosis (FALS) associated with superoxide dismutase 1 (SOD1) mutations, but axon-autonomous effects of pathogenic SOD1 remained undefine