Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Linda J. Zhu"'
Autor:
Shubha Murthy, Olha M Koval, Juan M Ramiro Diaz, Santosh Kumar, Daniel Nuno, Jason A Scott, Chantal Allamargot, Linda J Zhu, Kim Broadhurst, Velarchana Santhana, William J Kutschke, Kaikobad Irani, Kathryn G Lamping, Isabella M Grumbach
Publikováno v:
PLoS ONE, Vol 12, Iss 10, p e0186311 (2017)
The multifunctional Ca2+/calmodulin-dependent protein kinase II (CaMKII) is a serine/threonine kinase important in transducing intracellular Ca2+ signals. While in vitro data regarding the role of CaMKII in the regulation of endothelial nitric oxide
Externí odkaz:
https://doaj.org/article/f354a460347249199e1fb8733db21214
Autor:
Ellen Brinza, Heather L. Gornik, Michael P. Lilly, Laurence Needleman, Warren J. Manning, Linda J. Zhu
Publikováno v:
Journal for Vascular Ultrasound. 40:63-69
Objective Accreditation for noninvasive vascular testing facilities has been available since 1990, but is not mandatory. We sought to determine the perceived value of accreditation among the staff of vascular laboratories accredited by the Intersocie
Autor:
Chantal Allamargot, Kim Broadhurst, Isabella M. Grumbach, Velarchana Santhana, Daniel W. Nuno, William J. Kutschke, Kaikobad Irani, Jason A. Scott, Santosh Kumar, Shubha Murthy, Kathryn G. Lamping, Olha M. Koval, Linda J. Zhu, Juan M. Ramiro Diaz
Publikováno v:
PLoS ONE
PLoS ONE, Vol 12, Iss 10, p e0186311 (2017)
PLoS ONE, Vol 12, Iss 10, p e0186311 (2017)
The multifunctional Ca2+/calmodulin-dependent protein kinase II (CaMKII) is a serine/threonine kinase important in transducing intracellular Ca2+ signals. While in vitro data regarding the role of CaMKII in the regulation of endothelial nitric oxide
Autor:
Mark E. Anderson, Ashlee N. Venema, Isabella M. Grumbach, Elizabeth D. Luczak, Emily K. Nguyen, Megan E. Dibbern, Omar A. Jaffer, Xiaoqun Guan, Paula J. Klutho, Jason A. Scott, Linda J. Zhu, Anand Prasad, Litao Xie
Publikováno v:
Vascular pharmacology. 60(2)
Activation of the Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and reactive oxygen species (ROS) promote neointimal hyperplasia after vascular injury. CaMKII can be directly activated by ROS through oxidation. In this study, we determined w