Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Linda A Leis"'
Publikováno v:
PLoS ONE, Vol 11, Iss 6, p e0157855 (2016)
BACKGROUND:More than twenty years following the end of the 1990-1991 Gulf War it is estimated that approximately 300,000 veterans of this conflict suffer from an unexplained chronic, multi-system disorder known as Gulf War Illness (GWI). The etiology
Externí odkaz:
https://doaj.org/article/2a8265d1c5d441319570f114a4662319
Publikováno v:
PLoS ONE
PLoS ONE, Vol 11, Iss 6, p e0157855 (2016)
PLoS ONE, Vol 11, Iss 6, p e0157855 (2016)
Background More than twenty years following the end of the 1990–1991 Gulf War it is estimated that approximately 300,000 veterans of this conflict suffer from an unexplained chronic, multi-system disorder known as Gulf War Illness (GWI). The etiolo
Publikováno v:
Thrombosis and Haemostasis. 102:728-734
SummaryTissue factor (TF) is present in small quantities in normal blood and is reported to be elevated in arterial and venous thrombosis. Patients undergoing total knee arthoplasty (TKA) are at high risk of post-operative venous thromboembolism (VTE
Publikováno v:
The FASEB Journal. 28
As many as 200,000 veterans of the 1990-1991 Gulf War suffer from unexplained chronic multisymptom illnesses collectively known as Gulf War Illness (GWI). Symptoms of GWI may include muscle and joi...
Publikováno v:
American Journal of Physiology-Cell Physiology. 279:C1760-C1771
This study evaluated the mechanism of epinephrine potentiation of platelet secretion induced by thromboxane A2(TXA2). Dog platelets that do not secrete in response to TXA2alone (TXA2−) were compared with dog platelets that do secrete (TXA2+) and wi
Publikováno v:
Blood coagulationfibrinolysis : an international journal in haemostasis and thrombosis. 24(7)
A previous study of Gulf War veteran's illnesses (GWVI) observed evidence of platelet activation in a majority of patients with GWVI. To further characterize platelet function, we studied 43 patients (40 men) with GWVI (GWVI+) and 21 veterans who ser
Publikováno v:
Thrombosis Research. 59:139-148
Calcium channel antagonists inhibit platelet function in vitro and ex vivo , but the mechanism responsible has not been clearly defined. The concentrations of these agents required to inhibit platelet aggregation in vitro are several fold higher than
G alpha q and G alpha 11, members of the Gq family of G-proteins, transduce signals from receptors to the beta isoenzymes of phosphatidyl-inositol-specific phospholipase C (PI-PLC). The receptor specificity of these alpha subunits is unknown. G alpha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bf385520c4131b30a52a662e78cd6249
https://europepmc.org/articles/PMC1217719/
https://europepmc.org/articles/PMC1217719/
Publikováno v:
Blood. 55:722-729
Recent studies in our laboratory demonstrated that 30% of normal mongrel dogs have platelets that aggregate when stirred with sodium arachidonate (ASP), while the majority have platelets that do not aggregate under similar circumstances (AIP). In the
Publikováno v:
Thrombosis Research. 14:147-154
The platelet rich plasma (PRP) from most dogs aggregates in response to ADP, collagen and thrombin. Dog PRP generally does not aggregate in response to epinephrine, and previous studies found that dog PRP uniformly failed to aggregate when exposed to