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Autor:
Lincoln Shade, Erin Abner, Andrew Saykin, Timothy Hohman, Steven Claas, Shubhabrata Mukherjee, Mark Ebbert
BackgroundAlzheimer’s disease is highly heritable and exhibits neuropathological hallmarks of neurofibrillary tau tangles and neuritic amyloid plaques. Previous genome-wide association studies (GWAS) have identified over 70 genomic risk loci of cli
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::90cc14c4cad152099f3919c87e5415e1
https://doi.org/10.1101/2022.12.13.22282475
https://doi.org/10.1101/2022.12.13.22282475