Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Lina Abu-Tair"'
Publikováno v:
Cell Death Discovery, Vol 10, Iss 1, Pp 1-11 (2024)
Abstract Breast cancer is the leading cause of cancer-related deaths in women worldwide, with the basal-like or triple-negative breast cancer (TNBC) subtype being particularly aggressive and challenging to treat. Understanding the molecular mechanism
Externí odkaz:
https://doaj.org/article/fde5a074f8b34b6c86d4bd817fca586a
Autor:
Hussam Husanie, Muhannad Abu-Remaileh, Kian Maroun, Lina Abu-Tair, Hazem Safadi, Karine Atlan, Talia Golan, Rami I. Aqeilan
Publikováno v:
Cell Death and Disease, Vol 13, Iss 12, Pp 1-14 (2022)
Abstract Pancreatic cancer is one of the most lethal cancers, owing to its late diagnosis and resistance to chemotherapy. The tumor suppressor WW domain-containing oxidoreductase (WWOX), one of the most active fragile sites in the human genome (FRA16
Externí odkaz:
https://doaj.org/article/524624a72b364e70afa3e58a7f89ab8c
Autor:
Lina Abu-Tair, Jonathan H Axelrod, Sarit Doron, Yossi Ovadya, Valery Krizhanovsky, Eithan Galun, Johnny Amer, Rifaat Safadi
Publikováno v:
PLoS ONE, Vol 8, Iss 12, p e82571 (2013)
The toll-like receptor-9 (TLR9) agonist cytosine phosphate guanine (CpG), activates hepatic stellate cells (HSCs) and mediates fibrosis. We investigated the TLR9 effects on lymphocyte/HSCs interactions. Liver fibrosis was induced in wild-type (WT) mi
Externí odkaz:
https://doaj.org/article/dc94c8140179432990dd5cf30249f0e3
Autor:
Mahmud Mahamid, Nidal Muhanna, Maysa Azzeh, Johnny Amer, S. Doron, Lina Abu Tair, Scott L. Friedman, Rifaat Safadi
Publikováno v:
Gut. 60(1)
Background and aims Interactions between hepatic stellate cells (HSCs) and immune cell subsets have emerged as important determinants of liver fibrosis progression and regression. Natural killer (NK) cells have an antifibrotic activity through killin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5dd5039c12f7984827d009ba2badecb5
https://pubmed.ncbi.nlm.nih.gov/21398677
https://pubmed.ncbi.nlm.nih.gov/21398677