Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Lily E. Kisia"'
Autor:
Lily E. Kisia, Qiuying Cheng, Evans Raballah, Elly O. Munde, Benjamin H. McMahon, Nick W. Hengartner, John M. Ong’echa, Kiprotich Chelimo, Christophe G. Lambert, Collins Ouma, Prakasha Kempaiah, Douglas J. Perkins, Kristan A. Schneider, Samuel B. Anyona
Publikováno v:
Tropical Medicine and Health, Vol 50, Iss 1, Pp 1-15 (2022)
Abstract Plasmodium falciparum infections remain among the leading causes of morbidity and mortality in holoendemic transmission areas. Located within region 5q31.1, the colony-stimulating factor 2 gene (CSF2) encodes granulocyte–macrophage colony-
Externí odkaz:
https://doaj.org/article/9181a3a765424ab5950663800c7efad4
Autor:
Lily E. Kisia, Prakasha Kempaiah, Samuel B. Anyona, Elly O. Munde, Angela O. Achieng, John M. Ong’echa, Christophe G. Lambert, Kiprotich Chelimo, Collins Ouma, Douglas J. Perkins, Evans Raballah
Publikováno v:
BMC Medical Genetics, Vol 20, Iss 1, Pp 1-10 (2019)
Abstract Background Severe malarial anemia (SMA) is a leading cause of malaria-related morbidity and mortality in children. The genetic factors that influence development of SMA and inefficient erythropoiesis, a central pathogenic feature of SMA, are
Externí odkaz:
https://doaj.org/article/d64936deb1244e07876c59d122a1102c
Autor:
Samuel B. Anyona, Evans Raballah, Prakasha Kempaiah, Elly O. Munde, Kiprotich Chelimo, Angela O. Achieng, John M. Ong’echa, Douglas J. Perkins, Lily E. Kisia, Collins Ouma, Christophe G. Lambert
Publikováno v:
BMC Medical Genetics, Vol 20, Iss 1, Pp 1-10 (2019)
BMC Medical Genetics
BMC Medical Genetics
Background Severe malarial anemia (SMA) is a leading cause of malaria-related morbidity and mortality in children. The genetic factors that influence development of SMA and inefficient erythropoiesis, a central pathogenic feature of SMA, are only par
Autor:
Lily E. Kisia, Ng'wena G. Magak, Lyticia A Ochola, Collins Ouma, Estela Shabani, Cyrus Ayieko, Chandy C. John
Publikováno v:
Infection and Immunity. 82:3775-3782
Individuals naturally exposed to Plasmodium falciparum lose clinical immunity after a prolonged lack of exposure. P. falciparum antigen-specific cytokine responses have been associated with protection from clinical malaria, but the longevity of P. fa