Zobrazeno 1 - 10
of 42
pro vyhledávání: '"Lidia Ruiz-Llorente"'
Autor:
Miguel A. Ortega, Leonel Pekarek, Oscar Fraile-Martinez, Cielo Garcia-Montero, Miguel A. Saez, Angel Asúnsolo, Miguel A. Alvarez-Mon, Jorge Monserrat, Lidia Ruiz-Llorente, Natalio García-Honduvilla, Agustin Albillos, Julia Buján, Melchor Alvarez-Mon, Luis G. Guijarro
Publikováno v:
Current Oncology, Vol 29, Iss 4, Pp 2442-2453 (2022)
Pancreatic cancer will be positioned by the year 2030 as the second cause of oncological death after lung cancer. The pathophysiology of the most common variety, which involves the adenocarcinoma of the pancreas, represents one of the main challenges
Externí odkaz:
https://doaj.org/article/d0f169879a514bec891daaaf6c2c71fa
Autor:
Luis G. Guijarro, David Cano-Martínez, M.Val Toledo-Lobo, Patricia Sanmartín Salinas, María Chaparro, Ana M. Gómez-Lahoz, Sofía Zoullas, Rosa Rodríguez-Torres, Irene D. Román, Laura Sebastián Monasor, Lidia Ruiz-Llorente, María del Carmen Boyano-Adánez, Iván Guerra, Marisa Iborra, José Luis Cabriada, Luis Bujanda, Carlos Taxonera, Valle García-Sánchez, Ignacio Marín-Jiménez, Manuel Barreiro-de Acosta, Isabel Vera, María Dolores Martín-Arranz, Francisco Mesonero, Laura Sempere, Fernando Gomollón, Joaquín Hinojosa, Melchor Alvarez-Mon, Javier P. Gisbert, Miguel A. Ortega, Borja Hernández-Breijo, on behalf of the PREDICROHN study group from GETECCU
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 144, Iss , Pp 112239- (2021)
Inflammatory bowel diseases (IBD), represented by ulcerative colitis (UC) and Crohn's disease (CD), are characterized by chronic inflammation of the gastrointestinal tract, what leads to diarrhea, malnutrition, and weight loss. Depression of the grow
Externí odkaz:
https://doaj.org/article/07a4ccf32ca74a43a9db582d7fa13f0a
Autor:
Joshua Hodgson, Lidia Ruiz‐Llorente, Jamie McDonald, Oliver Quarrell, Kelechi Ugonna, James Bentham, Rebecca Mason, Jennifer Martin, David Moore, Katie Bergstrom, Pinar Bayrak‐Toydemir, Whitney Wooderchak‐Donahue, Nicholas W. Morrell, Robin Condliffe, Carmelo Bernabeu, Paul D. Upton
Publikováno v:
Molecular Genetics & Genomic Medicine, Vol 9, Iss 12, Pp n/a-n/a (2021)
Abstract Background Disrupted endothelial BMP9/10 signaling may contribute to the pathophysiology of both hereditary hemorrhagic telangiectasia (HHT) and pulmonary arterial hypertension (PAH), yet loss of circulating BMP9 has not been confirmed in in
Externí odkaz:
https://doaj.org/article/9256e773405946d6b2a4f5503805f3aa
Autor:
Luis G. Guijarro, David Cano-Martínez, M. Val Toledo-Lobo, Lidia Ruiz-Llorente, María Chaparro, Iván Guerra, Marisa Iborra, José Luis Cabriada, Luis Bujanda, Carlos Taxonera, Valle García-Sánchez, Ignacio Marín-Jiménez, Manuel Barreiro-de Acosta, Isabel Vera, María Dolores Martín-Arranz, Francisco Mesonero, Laura Sempere, Fernando Gomollón, Joaquín Hinojosa, Sofía Zoullas, Jorge Monserrat, Cesar Menor-Salvan, Melchor Alvarez-Mon, Javier P. Gisbert, Miguel A. Ortega, Borja Hernández-Breijo
Publikováno v:
Biomedicines, Vol 10, Iss 3, p 727 (2022)
Background: Recently, increased tissue levels of AIF-1 have been shown in experimental colitis, supporting its role in intestinal inflammation. Therefore, we studied the levels of AIF-1 in Crohn’s disease (CD). Methods: This study included 33 patie
Externí odkaz:
https://doaj.org/article/8729a017e24a4e2ca4c8aa824c8777c8
Autor:
Lidia Ruiz-Llorente, M. Cristina Vega, Francisco J. Fernández, Carmen Langa, Nicholas W. Morrell, Paul D. Upton, Carmelo Bernabeu
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 20, p 11282 (2021)
Endoglin (Eng, CD105) is a type I membrane glycoprotein that functions in endothelial cells as an auxiliary receptor for transforming growth factor β (TGF-β)/bone morphogenetic protein (BMP) family members and as an integrin ligand, modulating the
Externí odkaz:
https://doaj.org/article/600c446356be499ba3670854f6ecc105
Publikováno v:
Journal of Clinical Medicine, Vol 9, Iss 9, p 2855 (2020)
Hereditary hemorrhagic telangiectasia (HHT) is an autosomal dominant, vascular disorder that presents with telangiectases and arteriovenous malformations. HHT is a genetically heterogeneous disorder, involving mutations in endoglin (ENG; HHT1) and ac
Externí odkaz:
https://doaj.org/article/71bfec42196043b4b0d5ac3d95b1e359
Autor:
Eunate Gallardo-Vara, Lidia Ruiz-Llorente, Juan Casado-Vela, María J. Ruiz-Rodríguez, Natalia López-Andrés, Asit K. Pattnaik, Miguel Quintanilla, Carmelo Bernabeu
Publikováno v:
Cells, Vol 8, Iss 9, p 1082 (2019)
Endoglin is a 180-kDa glycoprotein receptor primarily expressed by the vascular endothelium and involved in cardiovascular disease and cancer. Heterozygous mutations in the endoglin gene (ENG) cause hereditary hemorrhagic telangiectasia type 1, a vas
Externí odkaz:
https://doaj.org/article/7ded8255c2cc400e8cc168fc3a811ab1
Autor:
Constanza Contreras-Jurado, Laura García-Serrano, Mónica Martínez-Fernández, Lidia Ruiz-Llorente, Jesus M Paramio, Ana Aranda
Publikováno v:
PLoS ONE, Vol 9, Iss 9, p e108137 (2014)
Both clinical and experimental observations show that the skin is affected by the thyroidal status. In hypothyroid patients the epidermis is thin and alopecia is common, indicating that thyroidal status might influence not only skin proliferation but
Externí odkaz:
https://doaj.org/article/3ed8e4e13de344af97c947ad056a33e8
Autor:
Lidia Ruiz-Llorente, María Jesús Ruiz-Rodríguez, Claudia Savini, Teresa González-Muñoz, Erica Riveiro-Falkenbach, José L. Rodríguez-Peralto, Héctor Peinado, Carmelo Bernabeu
Publikováno v:
Advances in Experimental Medicine and Biology ISBN: 9783031261626
20 p.-8 fig.-1 tab.
Endoglin (CD105) is an auxiliary receptor of transforming growth factor (TGF)-β family members that is expressed in human melanomas. It is heterogeneously expressed by primary and metastatic melanoma cells, and endoglin targ
Endoglin (CD105) is an auxiliary receptor of transforming growth factor (TGF)-β family members that is expressed in human melanomas. It is heterogeneously expressed by primary and metastatic melanoma cells, and endoglin targ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::879c2e9b1b92d8a8f19b0500b60fdb29
http://hdl.handle.net/10261/308227
http://hdl.handle.net/10261/308227
Autor:
Pinar Bayrak-Toydemir, Lidia Ruiz-Llorente, Oliver Quarrell, Katie Bergstrom, Jamie McDonald, Robin Condliffe, Nicholas W. Morrell, Kelechi Ugonna, Rebecca Mason, Jennifer M. Martin, David Moore, Carmelo Bernabeu, Paul D. Upton, Whitney Wooderchak-Donahue, Joshua Hodgson, James R. Bentham
Publikováno v:
Molecular Genetics & Genomic Medicine, Vol 9, Iss 12, Pp n/a-n/a (2021)
Molecular Genetics & Genomic Medicine
Digital.CSIC. Repositorio Institucional del CSIC
instname
Molecular Genetics & Genomic Medicine
Digital.CSIC. Repositorio Institucional del CSIC
instname
13 p.-3 fig.-1 tab.
Background: Disrupted endothelial BMP9/10 signaling may contribute to the pathophysiology of both hereditary hemorrhagic telangiectasia (HHT) and pulmonary arterial hypertension (PAH), yet loss of circulating BMP9 has not bee
Background: Disrupted endothelial BMP9/10 signaling may contribute to the pathophysiology of both hereditary hemorrhagic telangiectasia (HHT) and pulmonary arterial hypertension (PAH), yet loss of circulating BMP9 has not bee