Zobrazeno 1 - 10
of 30
pro vyhledávání: '"Lidia Lopez-Serra"'
Autor:
Rocio G Urdinguio, Lidia Lopez-Serra, Pilar Lopez-Nieva, Miguel Alaminos, Ramon Diaz-Uriarte, Agustin F Fernandez, Manel Esteller
Publikováno v:
PLoS ONE, Vol 3, Iss 11, p e3669 (2008)
BACKGROUND: Rett syndrome (RTT) is a complex neurological disorder that is one of the most frequent causes of mental retardation in women. A great landmark in research in this field was the discovery of a relationship between the disease and the pres
Externí odkaz:
https://doaj.org/article/f5f617b30532421da8549fe8ae2c94ee
Autor:
Marta Sánchez-Carbayo, Manel Esteller, Javier García del Muro, Javier García, Carlos Cordon-Cardo, Pilar Gonzalez-Peramato, Antonio López-Beltrán, Joaquin Bellmunt, Ferran Algaba, Lidia Lopez-Serra, Esteban Orenes, Virginia Lopez, Miguel Alvarez, Virginia Cebrian, Ainel Aleman
Supplementary Data from Identification of PMF1 Methylation in Association with Bladder Cancer Progression
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5981b9960495039ef369e7b386163c9e
https://doi.org/10.1158/1078-0432.22439616.v1
https://doi.org/10.1158/1078-0432.22439616.v1
Autor:
Marta Sánchez-Carbayo, Manel Esteller, Javier García del Muro, Javier García, Carlos Cordon-Cardo, Pilar Gonzalez-Peramato, Antonio López-Beltrán, Joaquin Bellmunt, Ferran Algaba, Lidia Lopez-Serra, Esteban Orenes, Virginia Lopez, Miguel Alvarez, Virginia Cebrian, Ainel Aleman
Purpose: Polyamines are important regulators of cell growth and death. The polyamine modulated factor-1 (PMF-1) is involved in polyamine homeostasis. After identifying an enriched CpG island encompassing the PMF1 promoter, we aimed at evaluating the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a0295441061c0167ec71a5e5c969260
https://doi.org/10.1158/1078-0432.c.6517056.v1
https://doi.org/10.1158/1078-0432.c.6517056.v1
Autor:
Manel Esteller, Fernando Setien, Miguel Alaminos, Mario F. Fraga, Esteban Ballestar, Lidia Lopez-Serra
Methyl-CpG binding domain (MBD) proteins have been shown to couple DNA methylation to transcriptional repression. This biological property suggests a role for MBD proteins in the silencing of tumor suppressor genes that are hypermethylated at their p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5f1a658bed5467dda4b2630e4a0e65c7
https://doi.org/10.1158/0008-5472.c.6494480
https://doi.org/10.1158/0008-5472.c.6494480
Autor:
Manel Esteller, Fernando Setien, Miguel Alaminos, Mario F. Fraga, Esteban Ballestar, Lidia Lopez-Serra
Supplementary Table 2 from A Profile of Methyl-CpG Binding Domain Protein Occupancy of Hypermethylated Promoter CpG Islands of Tumor Suppressor Genes in Human Cancer
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0726b502f43d673af3310a1f12350065
https://doi.org/10.1158/0008-5472.22364999.v1
https://doi.org/10.1158/0008-5472.22364999.v1
Autor:
Manel Esteller, Fernando Setien, Miguel Alaminos, Mario F. Fraga, Esteban Ballestar, Lidia Lopez-Serra
Supplementary Table 1 from A Profile of Methyl-CpG Binding Domain Protein Occupancy of Hypermethylated Promoter CpG Islands of Tumor Suppressor Genes in Human Cancer
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2dea1b47abcd1d7c713246882998a68d
https://doi.org/10.1158/0008-5472.22365002
https://doi.org/10.1158/0008-5472.22365002
Publikováno v:
Nature Genetics
Nature genetics
Nature genetics
The cohesin complex is at the heart of many chromosomal activities, including sister chromatid cohesion and transcriptional regulation. Cohesin loading onto chromosomes depends on the Scc2-Scc4 cohesin loader complex, but the chromatin features that
Publikováno v:
Yeast. 29:233-240
The ease with which populations of the budding yeast Saccharomyces cerevisiae can be synchronized using the mating pheromone α-factor has been invaluable for studies of the cell cycle. The α-factor response pathway has also remained an important mo
Autor:
Elisa Carrasco, Fernando Setien, Manel Esteller, Lidia Lopez-Serra, Paula Lopez-Serra, Héctor Peinado, Jaime Renart, Amparo Cano, María I. Calvo, Anna Portela, Angeles Juarranz, Jesús Espada
Publikováno v:
Nucleic Acids Research; Vol 39
Digital.CSIC. Repositorio Institucional del CSIC
instname
Recercat. Dipósit de la Recerca de Catalunya
Nucleic Acids Research
Biblos-e Archivo. Repositorio Institucional de la UAM
Dipòsit Digital de la UB
Universidad de Barcelona
Digital.CSIC. Repositorio Institucional del CSIC
instname
Recercat. Dipósit de la Recerca de Catalunya
Nucleic Acids Research
Biblos-e Archivo. Repositorio Institucional de la UAM
Dipòsit Digital de la UB
Universidad de Barcelona
This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License.-- et al.
Mammalian DNA methyltransferase 1 (DNMT1) is essential for maintaining DNA methylation patterns after cell division.
Mammalian DNA methyltransferase 1 (DNMT1) is essential for maintaining DNA methylation patterns after cell division.
Autor:
José I, Martín-Subero, Markus, Kreuz, Marina, Bibikova, Stefan, Bentink, Ole, Ammerpohl, Eliza, Wickham-Garcia, Maciej, Rosolowski, Julia, Richter, Lidia, Lopez-Serra, Esteban, Ballestar, Hilmar, Berger, Xabier, Agirre, Heinz-Wolfram, Bernd, Vincenzo, Calvanese, Sergio B, Cogliatti, Hans G, Drexler, Jian-Bing, Fan, Mario F, Fraga, Martin L, Hansmann, Michael, Hummel, Wolfram, Klapper, Bernhard, Korn, Ralf, Küppers, Roderick A F, Macleod, Peter, Möller, German, Ott, Christiane, Pott, Felipe, Prosper, Andreas, Rosenwald, Carsten, Schwaenen, Dirk, Schübeler, Marc, Seifert, Benjamin, Stürzenhofecker, Michael, Weber, Swen, Wessendorf, Markus, Loeffler, Lorenz, Trümper, Harald, Stein, Rainer, Spang, Manel, Esteller, David, Barker, Dirk, Hasenclever, Reiner, Siebert, Maren, Wehner
Publikováno v:
Blood
Blood, American Society of Hematology, 2009, 113 (11), pp.2488-2497. ⟨10.1182/blood-2008-04-152900⟩
Blood, American Society of Hematology, 2009, 113 (11), pp.2488-2497. ⟨10.1182/blood-2008-04-152900⟩
Lymphomas are assumed to originate at different stages of lymphocyte development through chromosomal aberrations. Thus, different lymphomas resemble lymphocytes at distinct differentiation stages and show characteristic morphologic, genetic, and tran