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Publikováno v:
Pharmacological Research, Vol 203, Iss , Pp 107165- (2024)
The clinical use of the DNA damaging anticancer drug doxorubicin (DOX) is limited by irreversible cardiotoxicity, which depends on the cumulative dose. The RAS-homologous (RHO) small GTPase RAC1 contributes to DOX-induced DNA damage formation and car
Externí odkaz:
https://doaj.org/article/895091e38441435ab22406d0afdc7869