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α7-Nicotinic Acetylcholine Receptor Agonist Ameliorates Nicotine Plus High-Fat Diet-Induced Hepatic Steatosis in Male Mice by Inhibiting Oxidative Stress and Stimulating AMPK Signaling

Autor: Hasan, Mohammad Kamrul, Friedman, Theodore C, Sims, Carl, Lee, Desean L, Espinoza-Derout, Jorge, Ume, Adaku, Chalfant, Victor, Lee, Martin L, Sinha-Hikim, Indrani, Lutfy, Kabirullah, Liu, Yanjun, Mahata, Sushil K, Sinha-Hikim, Amiya P

Publikováno v: Endocrinology, vol 159, iss 2

α7-Nicotinic acetylcholine receptor (α7nAChR) agonists confer protection against a wide variety of cytotoxic insults and suppress oxidative stress and apoptosis in various cell systems, including hepatocytes. We recently demonstrated that nicotine,

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::28244973c6137f844523d5b07b283353
https://escholarship.org/uc/item/9qc962kh

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α7-Nicotinic Acetylcholine Receptor Agonist Ameliorates Nicotine Plus High-Fat Diet-Induced Hepatic Steatosis in Male Mice by Inhibiting Oxidative Stress and Stimulating AMPK Signaling.

Autor: Hasan MK; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California., Friedman TC; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California.; David Geffen School of Medicine at University of California, Los Angeles, Los Angeles, California., Sims C; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California., Lee DL; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California., Espinoza-Derout J; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California., Ume A; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California., Chalfant V; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California., Lee ML; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California.; David Geffen School of Medicine at University of California, Los Angeles, Los Angeles, California., Sinha-Hikim I; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California.; David Geffen School of Medicine at University of California, Los Angeles, Los Angeles, California., Lutfy K; College of Pharmacy, Western University of Health Sciences, Pomona, California., Liu Y; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California., Mahata SK; Department of Medicine, University of California, San Diego, San Diego, California.; VA San Diego Health Care System, San Diego, California., Sinha-Hikim AP; Division of Endocrinology, Metabolism and Molecular Medicine, Department of Internal Medicine, Charles R. Drew University of Medicine and Science, Los Angeles, California.; David Geffen School of Medicine at University of California, Los Angeles, Los Angeles, California.

Publikováno v: Endocrinology [Endocrinology] 2018 Feb 01; Vol. 159 (2), pp. 931-944.