Zobrazeno 1 - 10
of 41
pro vyhledávání: '"Lechado-Terradas A"'
Autor:
Daniel J. Merk, Foteini Tsiami, Sophie Hirsch, Bianca Walter, Lara A. Haeusser, Jens D. Maile, Aaron Stahl, Mohamed A. Jarboui, Anna Lechado-Terradas, Franziska Klose, Sepideh Babaei, Jakob Admard, Nicolas Casadei, Cristiana Roggia, Michael Spohn, Jens Schittenhelm, Stephan Singer, Ulrich Schüller, Federica Piccioni, Nicole S. Persky, Manfred Claassen, Marcos Tatagiba, Philipp J. Kahle, David E. Root, Markus Templin, Ghazaleh Tabatabai
Publikováno v:
Genome Biology, Vol 25, Iss 1, Pp 1-38 (2024)
Abstract Background Atypical teratoid rhabdoid tumors (ATRT) are incurable high-grade pediatric brain tumors. Despite intensive research efforts, the prognosis for ATRT patients under currently established treatment protocols is poor. While novel the
Externí odkaz:
https://doaj.org/article/c3f097f8f86b4c4a8b8f3ec94c994ed5
Autor:
Merk, Daniel J., Tsiami, Foteini, Hirsch, Sophie, Walter, Bianca, Haeusser, Lara A., Maile, Jens D., Stahl, Aaron, Jarboui, Mohamed A., Lechado-Terradas, Anna, Klose, Franziska, Babaei, Sepideh, Admard, Jakob, Casadei, Nicolas, Roggia, Cristiana, Spohn, Michael, Schittenhelm, Jens, Singer, Stephan, Schüller, Ulrich, Piccioni, Federica, Persky, Nicole S.
Publikováno v:
Genome Biology; 12/2/2024, Vol. 25 Issue 1, p1-38, 38p
Autor:
Sharma K; Department of Neurodegeneration, Hertie Institute for Clinical Brain Research, University of Tübingen, Tübingen, Germany., Kishore A; Department of Neurology, Sree Chitra Tirunal Institute for Medical Sciences and Technology, Thiruvananthapuram, India.; Parkinson and Movement Disorder Centre, Aster Medicity, Kochi, India., Lechado-Terradas A; Department of Neurodegeneration, Hertie Institute for Clinical Brain Research, University of Tübingen, Tübingen, Germany., Passannanti R; Laboratorio di Biologia Bio@SNS, Scuola Normale Superiore, Pisa, Italy., Raimondi F; Laboratorio di Biologia Bio@SNS, Scuola Normale Superiore, Pisa, Italy., Sturm M; Institute of Medical Genetics and Applied Genomics, University of Tübingen, Tübingen, Germany., Sreelatha AAK; Centre for Genetic Epidemiology, Institute for Clinical Epidemiology and Applied Biometry, University of Tübingen, Tübingen, Germany., Puthenveedu DK; Department of Neurology, Sree Chitra Tirunal Institute for Medical Sciences and Technology, Thiruvananthapuram, India., Sarma G; Department of Neurology, Sree Chitra Tirunal Institute for Medical Sciences and Technology, Thiruvananthapuram, India., Casadei N; Institute of Medical Genetics and Applied Genomics & Core Facility for Applied Genomics, University of Tübingen, Tübingen, Germany., Krüger R; Translational Neuroscience, Luxembourg Center for Systems Biomedicine, University of Luxembourg, Luxembourg, Transversal Translational Medicine, Luxembourg Institute of Health, Strassen, Luxembourg & Centre Hospitalier de Luxembourg, Esch-sur-Alzette, Luxembourg., Gasser T; Department of Neurodegeneration, Hertie Institute for Clinical Brain Research, University of Tübingen, Tübingen, Germany.; German Centre for Neurodegenerative Diseases, Tübingen, Germany., Kahle P; Department of Neurodegeneration, Hertie Institute for Clinical Brain Research, University of Tübingen, Tübingen, Germany.; German Centre for Neurodegenerative Diseases, Tübingen, Germany.; Department of Biochemistry, University of Tübingen, Tübingen, Germany., Riess O; Institute of Medical Genetics and Applied Genomics & Core Facility for Applied Genomics, University of Tübingen, Tübingen, Germany., Fitzgerald JC; Department of Neurodegeneration, Hertie Institute for Clinical Brain Research, University of Tübingen, Tübingen, Germany., Sharma M; Centre for Genetic Epidemiology, Institute for Clinical Epidemiology and Applied Biometry, University of Tübingen, Tübingen, Germany.
Publikováno v:
Movement disorders : official journal of the Movement Disorder Society [Mov Disord] 2024 Jul; Vol. 39 (7), pp. 1217-1225. Date of Electronic Publication: 2024 Apr 08.
Autor:
Anna Lechado-Terradas, Sandra Schepers, Katharina I. Zittlau, Karan Sharma, Orkun Ok, Julia C. Fitzgerald, Stefan Geimer, Benedikt Westermann, Boris Macek, Philipp J. Kahle
Publikováno v:
Autophagy Reports, Vol 1, Iss 1, Pp 576-602 (2022)
PINK1/parkin-dependent mitophagy initially involves (phospho)ubiquitin-directed proteasome-dependent degradation of certain outer mitochondrial membrane (OMM) proteins (e.g. mitofusins) and the recruitment of autophagy adaptors to a group of ubiquiti
Externí odkaz:
https://doaj.org/article/28921611ba294240a516a15b1a04f119
Autor:
Zittlau, Katharina I., Lechado-Terradas, Anna, Nalpas, Nicolas, Geisler, Sven, Kahle, Philipp J., Macek, Boris
Publikováno v:
In Molecular & Cellular Proteomics February 2022 21(2)
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Autor:
Lechado Terradas, Anna, Zittlau, Katharina I., Macek, Boris, Fraiberg, Milana, Elazar, Zvulun, Kahle, Philipp J.
Publikováno v:
In Journal of Biological Chemistry November 2021 297(5)
Autor:
Katharina I. Zittlau, Anna Lechado-Terradas, Nicolas Nalpas, Sven Geisler, Philipp J. Kahle, Boris Macek
Publikováno v:
Molecular & cellular proteomics 21(2), 100191 (2022). doi:10.1016/j.mcpro.2021.100191
Mitophagy, the selective degradation of mitochondria by autophagy, affects defective mitochondria following damage or stress. At the onset of mitophagy, parkin ubiquitylates proteins on the mitochondrial outer membrane. While the role of parkin at th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c42e55de34e838f8f0835150c07ca2ba
https://pub.dzne.de/record/163264
https://pub.dzne.de/record/163264
Autor:
Milana Fraiberg, Boris Macek, Zvulun Elazar, Philipp J. Kahle, Anna Lechado Terradas, Katharina Zittlau
Publikováno v:
The Journal of Biological Chemistry
The journal of biological chemistry 297(5), 101339 (2021). doi:10.1016/j.jbc.2021.101339
The journal of biological chemistry 297(5), 101339 (2021). doi:10.1016/j.jbc.2021.101339
Mitochondria are important organelles in eukaryotes. Turnover and quality control of mitochondria are regulated at the transcriptional and posttranslational level by several cellular mechanisms. Removal of defective mitochondrial proteins is mediated
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3fbec8d603702bfc18179f4f97b4c81b
http://europepmc.org/articles/PMC8591368
http://europepmc.org/articles/PMC8591368
Autor:
Anna Lechado Terradas, Friederike Hans, Sonia Golombek, Sven Geisler, Caren Linnemann, Etsuro Nakanishi, Lea Jäger, Nicolas Casadei, Philipp J. Kahle
Publikováno v:
Experimental cell research 384(2), 111641 (2019). doi:10.1016/j.yexcr.2019.111641
Parkin is an ubiquitin ligase regulating mitochondrial quality control reactions, including the autophagic removal of depolarized mitochondria (mitophagy). Parkin-mediated protein ubiquitinations may be counteracted by deubiquitinating enzymes (DUBs)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9699e494c46184a2a3abb8b9e13a1c01
https://doi.org/10.1016/j.yexcr.2019.111641
https://doi.org/10.1016/j.yexcr.2019.111641