Zobrazeno 1 - 10
of 24
pro vyhledávání: '"Lea Ziporen"'
Autor:
Yehuda Shoenfeld, Juri Kopolovic, D. Amos Korczyn, Joab Chapman, Iris Goldberg, Lea Ziporen, Arnon Afek, Sylvia Polak-Charcon
Publikováno v:
Clinical and Developmental Immunology, Vol 11, Iss 1, Pp 67-75 (2004)
Externí odkaz:
https://doaj.org/article/a5b212d71d0f4ac2883bb9c6545aab85
Autor:
Zvi Fishelson, Noam Shomron, Tamar Geiger, Lea Ziporen, Metsada Pasmanik-Chor, Mariya Mardamshina, Yaron Hillman
Supplementary Array S1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7fd620f82737e7d50e83e1f47de759fa
https://doi.org/10.1158/2326-6066.22542753
https://doi.org/10.1158/2326-6066.22542753
Autor:
Zvi Fishelson, Noam Shomron, Tamar Geiger, Lea Ziporen, Metsada Pasmanik-Chor, Mariya Mardamshina, Yaron Hillman
MicroRNAs (miR) are small RNA molecules that shape the cell transcriptome and proteome through regulation of mRNA stability and translation. Here, we examined their function as determinants of cell resistance to complement-dependent cytotoxicity (CDC
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7fc56474a666c753efad3bf187c43283
https://doi.org/10.1158/2326-6066.c.6550038
https://doi.org/10.1158/2326-6066.c.6550038
Autor:
Zvi Fishelson, Osnat Bairey, Ritta Jubran, Natalie Donin, Moran Saar-Ray, Anna Wawruszak, Lea Ziporen
Publikováno v:
International Journal of Oncology.
Cancer cells have developed numerous strategies to maintain their proliferative capacity and to withstand different kinds of stress. The mitochondrial stress‑70 protein named glucose regulated protein 75 (GRP75), also known as mortalin, is an intri
Autor:
Lea Ziporen, Zvi Fishelson, Mariya Mardamshina, Metsada Pasmanik-Chor, Noam Shomron, Yaron Hillman, Tamar Geiger
Publikováno v:
Cancer immunology research. 7(12)
MicroRNAs (miR) are small RNA molecules that shape the cell transcriptome and proteome through regulation of mRNA stability and translation. Here, we examined their function as determinants of cell resistance to complement-dependent cytotoxicity (CDC
Cooperation between Hsp90 and mortalin/GRP75 in resistance to cell death induced by complement C5b-9
Publikováno v:
Cell Death & Disease
Cell Death and Disease, Vol 9, Iss 2, Pp 1-11 (2018)
Cell Death and Disease, Vol 9, Iss 2, Pp 1-11 (2018)
Cancer cells are commonly more resistant to cell death activated by the membranolytic protein complex C5b-9. Several surface-expressed and intracellular proteins that protect cells from complement-dependent cytotoxicity (CDC) have been identified. In
Publikováno v:
The Journal of Immunology. 182:515-521
The membrane attack complex (MAC) of the complement system induces a necrotic-type cell death. Earlier findings suggested that Bcl-2 protects cells from MAC-induced necrosis. Here we examined the involvement of Bid, a proapoptotic protein, in MAC-ind
Autor:
Sylvia Polak-Charcon, D. Amos Korczyn, Joab Chapman, Juri Kopolovic, Lea Ziporen, Iris Goldberg, Arnon Afek, Yehuda Shoenfeld
Publikováno v:
Clinical and Developmental Immunology
Clinical and Developmental Immunology, Vol 11, Iss 1, Pp 67-75 (2004)
Clinical and Developmental Immunology, Vol 11, Iss 1, Pp 67-75 (2004)
The aim of this work was to study the pathological processes underlying neurological dysfunctions displayed by BALB/C mice induced with experimental antiphospholipid syndrome (APS), as we have previously reported. Experimental APS was induced in fema
Autor:
Katrin Jurianz, Michael Kirschfink, Natalie Donin, Lea Ziporen, Zvi Fishelson, Stefan Schultz
Publikováno v:
Clinical and Experimental Immunology. 131:254-263
SUMMARY Nucleated cells employ several strategies to evade killing by homologous complement. We studied complement resistance in the human carcinoma cell lines (CA) T47D (mammary), SKOV3 (ovarian), and PC-3 (prostate) with emphasis on the following m
Autor:
Arnon Afek, Juri Kopolovic, R Garcia-Torres, R Segal, Jacob George, M.C. Amigo, Lea Ziporen, Sylvie Polak-Charcon, R Manor, Y Shoenfeld, Iris Goldberg
Publikováno v:
Lupus. 8:502-507
The objective of this work was to determine markers of endothelial cell activation in valves from patients with antiphospholipid syndrome (APS) and heart valve involvement, in order to establish a role for endothelial cells in the pathogenesis of the