Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Laurence Tardif"'
Publikováno v:
Molecular Oncology, Vol 15, Iss 8, Pp 2106-2119 (2021)
The PI3K/Akt signaling pathway, the most frequently altered signaling system in human cancer, is a crucial inducer of dysregulated proliferation and neoplastic processes; however, few therapeutic strategies using PI3K/Akt inhibitors singly have been
Externí odkaz:
https://doaj.org/article/ff97c07ff94349dea3d0a80534f27624
Autor:
François Fabi, Kathy Grenier, Sophie Parent, Pascal Adam, Laurence Tardif, Valérie Leblanc, Eric Asselin
Publikováno v:
PLoS ONE, Vol 12, Iss 5, p e0177387 (2017)
Infertility is constantly increasing in Canada, where 16% of Canadian couples are experiencing difficulty conceiving. It is thought that infertility can emanate from the dysregulated communication between the embryo and the maternal endometrium. In o
Externí odkaz:
https://doaj.org/article/090871a051ce432e99ca42a0e5ac3ba2
Autor:
Pascal Adam, Laurence Tardif, François Fabi, Dadou Likonza Lokengo, Léa-Isabelle Renaud, Sophie Parent, Monique Cadrin, Derek Boerboom, Eric Asselin
Publikováno v:
Reproduction. 165:605-616
In brief The regulation of AKT in the endometrium during many cellular processes such as apoptosis and cell survival is crucial during the estrous cycle to ensure fertility. This research shows the specific function of AKT isoforms in the mouse endom
Autor:
Eric Asselin, François Fabi, Sophie Parent, Laurence Tardif, Kathy Grenier, Pascal Adam, Valérie Leblanc
Publikováno v:
PLoS ONE
PLoS ONE, Vol 12, Iss 5, p e0177387 (2017)
PLoS ONE, Vol 12, Iss 5, p e0177387 (2017)
Infertility is constantly increasing in Canada, where 16% of Canadian couples are experiencing difficulty conceiving. It is thought that infertility can emanate from the dysregulated communication between the embryo and the maternal endometrium. In o
Publikováno v:
Cancer Research. 76:1994-1994
Prostate Apoptosis Response-4 (Par-4) is a tumor suppressor protein whose expression level in cancer is frequently decreased; however, it is rarely mutated nor suppressed. Par-4 is not only regulated at the expression level, but also post-translation