Zobrazeno 1 - 10
of 66
pro vyhledávání: '"Lauren Peacock"'
Autor:
Lucinda R. Lee, Lauren Peacock, Leszek Lisowski, David G. Little, Craig F. Munns, Aaron Schindeler
Publikováno v:
Molecular Therapy: Methods & Clinical Development, Vol 15, Iss , Pp 101-111 (2019)
A panel of 18 recombinant adeno-associated virus (rAAV) variants, both natural and engineered, constitutively expressing Cre recombinase under the cytomegalovirus early enhancer/chicken β actin (CAG) promoter, were screened for their ability to tran
Externí odkaz:
https://doaj.org/article/b338fc69a1324de19ac8a0e1cd433523
Autor:
Roxanna J. Nahvi, Arax Tanelian, Chiso Nwokafor, Callie M. Hollander, Lauren Peacock, Esther L. Sabban
Publikováno v:
Frontiers in Behavioral Neuroscience, Vol 15 (2021)
The susceptibility to stress-elicited disorders is markedly influenced by sex. Women are twice as likely as men to develop posttraumatic stress disorder (PTSD), depression, anxiety disorders, and social impairments following exposure to traumatic str
Externí odkaz:
https://doaj.org/article/1adc130baa1d4734a86b3972f5b32c7b
Autor:
Michele Casanova, Aaron Schindeler, Lauren Peacock, Lucinda Lee, Philipp Schneider, David G. Little, Ralph Müller
Publikováno v:
JBMR Plus, Vol 5, Iss 9, Pp n/a-n/a (2021)
ABSTRACT Fracture repair is a normal physiological response to bone injury. During the process of bony callus formation, a lacunocanalicular network (LCN) is formed de novo that evolves with callus remodeling. Our aim was the longitudinal assessment
Externí odkaz:
https://doaj.org/article/0feeebd7f116472289932ebeef70e9a6
Autor:
David G. Little, Aaron Schindeler, Leszek Lisowski, Lauren Peacock, Craig F Munns, Lucinda R. Lee
Publikováno v:
Molecular Therapy: Methods & Clinical Development, Vol 15, Iss, Pp 101-111 (2019)
Molecular Therapy. Methods & Clinical Development
Molecular Therapy. Methods & Clinical Development
A panel of 18 recombinant adeno-associated virus (rAAV) variants, both natural and engineered, constitutively expressing Cre recombinase under the cytomegalovirus early enhancer/chicken β actin (CAG) promoter, were screened for their ability to tran
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7de620ad324cad1174582edc1cdb2ffd
http://www.sciencedirect.com/science/article/pii/S2329050119300907
http://www.sciencedirect.com/science/article/pii/S2329050119300907
Autor:
Ralph Müller, Philipp Schneider, Michele Casanova, David G. Little, Lauren Peacock, Lucinda R. Lee, Aaron Schindeler
Publikováno v:
JBMR Plus, 5 (9)
JBMR Plus, Vol 5, Iss 9, Pp n/a-n/a (2021)
JBMR Plus
JBMR Plus, Vol 5, Iss 9, Pp n/a-n/a (2021)
JBMR Plus
Fracture repair is a normal physiological response to bone injury. During the process of bony callus formation, a lacunocanalicular network (LCN) is formed de novo that evolves with callus remodeling. Our aim was the longitudinal assessment of the de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f0ff6b3026c6775efc3f21d4ac0531e
https://doi.org/10.1002/jbm4.10525
https://doi.org/10.1002/jbm4.10525
Autor:
Laurence C. Cantrill, Aaron Schindeler, Tegan L. Cheng, Lucinda R. Lee, Samantha L. Ginn, Craig F Munns, David G. Little, Lauren Peacock
Publikováno v:
Calcified Tissue International. 104:426-436
Bone marrow transplantation (BMT) of healthy donor cells has been postulated as a strategy for treating osteogenesis imperfecta (OI) and other bone fragility disorders. The effect of engraftment by tail vein injection and/or marrow ablation by 6 Gy w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::320854c9af0385c4576ca564e199da80
https://doi.org/10.1007/s00223-018-0504-3
https://doi.org/10.1007/s00223-018-0504-3
Autor:
Nikita Deo, David G. Little, Jad El-Hoss, Aaron Schindeler, Lauren Peacock, Mille Kolind, Kathy Mikulec
Publikováno v:
Journal of Applied Biomedicine. 16:350-357
Tibial pseudarthrosis often features deficient bone formation, excessive bone resorption, and extensive pathological fibrosis, particularly in individuals with Neurofibromatosis type I (NF1). It was hypothesized that overactive NF1-Ras-JNK signalling
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::301440ed6a962364113f9b1f9f01d54b
https://doi.org/10.1016/j.jab.2018.01.006
https://doi.org/10.1016/j.jab.2018.01.006
Autor:
Kathy Mikulec, Lauren Peacock, Emily R Vasiljevski, David G. Little, Aaron Schindeler, Matthew A. Summers
Publikováno v:
Molecular Genetics and Metabolism. 123:518-525
Neurofibromatosis Type 1 (NF1) is a common autosomal dominant genetic disorder While NF1 is primarily associated with predisposition for tumor formation, muscle weakness has emerged as having a significant impact on quality of life. NF1 inactivation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f1aeab48808d93fe41b20fa089f60447
https://doi.org/10.1016/j.ymgme.2018.02.009
https://doi.org/10.1016/j.ymgme.2018.02.009
Autor:
Sandra T. Cooper, David A. Stevenson, Kate G. R. Quinlan, Ute Roessner, Aaron Schindeler, Thusitha T.W. Rupasinghe, David G. Little, Emily R Vasiljevski, Kathy Mikulec, Matthew A. Summers, Lauren Peacock, Frances J. Evesson
Publikováno v:
Human Molecular Genetics. 27:577-588
Neurofibromatosis type 1 (NF1) is an autosomal dominant genetic disorder with complex symptomology. In addition to a predisposition to tumors, children with NF1 can present with reduced muscle mass, global muscle weakness, and impaired motor skills,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e0e831c8e9bbac43cf440366855af6e2
https://doi.org/10.1093/hmg/ddx423
https://doi.org/10.1093/hmg/ddx423