Zobrazeno 1 - 10
of 23
pro vyhledávání: '"Lauren M. Brown"'
Autor:
Stien De Coninck, Renate De Smedt, Beatrice Lintermans, Lindy Reunes, Hansen J. Kosasih, Alexandra Reekmans, Lauren M. Brown, Nadine Van Roy, Bruno Palhais, Juliette Roels, Malaika Van der Linden, Jo Van Dorpe, Panagiotis Ntziachristos, Frederik W. van Delft, Marc R. Mansour, Tim Pieters, Tim Lammens, Barbara De Moerloose, Charles E. de Bock, Steven Goossens, Pieter Van Vlierberghe
Publikováno v:
Haematologica, Vol 109, Iss 5 (2023)
T-cell acute lymphoblastic leukemia (T-ALL) and T-cell lymphoblastic lymphoma (T-LBL) are rare aggressive hematologic malignancies. Current treatment consists of intensive chemotherapy leading to 80% overall survival but is associated with severe tox
Externí odkaz:
https://doaj.org/article/a08f66779ce943b3bf87b6b00741dfb3
Autor:
Juan Fontana, Doina Atanasiu, Wan Ting Saw, John R. Gallagher, Reagan G. Cox, J. Charles Whitbeck, Lauren M. Brown, Roselyn J. Eisenberg, Gary H. Cohen
Publikováno v:
mBio, Vol 8, Iss 4 (2017)
ABSTRACT All enveloped viruses, including herpesviruses, must fuse their envelope with the host membrane to deliver their genomes into target cells, making this essential step subject to interference by antibodies and drugs. Viral fusion is mediated
Externí odkaz:
https://doaj.org/article/bedf0d56e13a4379a5a712758525c86a
Autor:
Lauren M. Brown
Publikováno v:
Studies in the Novel. 55:37-57
Autor:
Teresa Sadras, Fatimah B. Jalud, Hansen J. Kosasih, Christopher R. Horne, Lauren M. Brown, Sam El-Kamand, Charles E. de Bock, Lachlan McAloney, Ashley P. Ng, Nadia M. Davidson, Louise E. A. Ludlow, Alicia Oshlack, Mark J. Cowley, Seong L. Khaw, James M. Murphy, Paul G. Ekert
Publikováno v:
Leukemia. 37:905-909
Autor:
Breon Schmidt, Lauren M. Brown, Georgina L. Ryland, Andrew Lonsdale, Hansen J. Kosasih, Louise E. Ludlow, Ian J. Majewski, Piers Blombery, Paul G. Ekert, Nadia M. Davidson, Alicia Oshlack
Publikováno v:
Blood Advances. 6:4093-4097
Publikováno v:
Oncogene.
Rare but recurrent mutations in the fibroblast growth factor receptor (FGFR) pathways, most commonly in one of the four FGFR receptor tyrosine kinase genes, can potentially be targeted with broad-spectrum multi-kinase or FGFR selective inhibitors. Th
Autor:
Allen Gu, Breon Schmidt, Andrew Lonsdale, Lauren M. Brown, Teresa Sadras, Paul G. Ekert, Alicia Oshlack
T-cell acute lymphoblastic leukaemia (T-ALL) is an aggressive and heterogenous haematological malignancy affecting both children and adults. T-ALL subtype identification is an emerging area of active research, with several recent studies proposing po
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3148a38a9db5692d4dc27c25fb2bdd79
https://doi.org/10.1101/2023.04.05.535648
https://doi.org/10.1101/2023.04.05.535648
Autor:
Lauren M Brown
Publikováno v:
MELUS. 46:14-36
Toni Morrison’s 1999 book The Big Box, coauthored with her son, Slade Morrison, depicts three children who are locked up because they “just can’t handle [their] freedom.” Ostensibly written for children, the text nonetheless engages issues of
Autor:
Andrew Lonsdale, Andreas Halman, Lauren M Brown, Hansen J Kosasih, Paul G Ekert, Alicia Oshlack
Cancer is driven by mutations of the genome that can result in the activation of oncogenes or repression of tumour suppressor genes. In acute lymphoblastic leukemia (ALL) focal deletions in IKAROS family zinc finger 1 (IKZF1) result in the loss of zi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a420b766eb07d534262b7651dc4b73f3
https://doi.org/10.1101/2022.10.27.514132
https://doi.org/10.1101/2022.10.27.514132
SFPQ-ABL1 and BCR-ABL1 use different signaling networks to drive B-cell acute lymphoblastic leukemia
Autor:
Lauren M. Brown, Soroor Hediyeh-zadeh, Teresa Sadras, Hannah Huckstep, Jarrod J. Sandow, Ray C. Bartolo, Hansen J. Kosasih, Nadia M. Davidson, Breon Schmidt, Stefan Bjelosevic, Ricky Johnstone, Andrew I. Webb, Seong L. Khaw, Alicia Oshlack, Melissa J. Davis, Paul G. Ekert
Publikováno v:
Blood advances. 6(7)
Philadelphia-like (Ph-like) acute lymphoblastic leukemia (ALL) is a high-risk subtype of B-cell ALL characterized by a gene expression profile resembling Philadelphia chromosome–positive ALL (Ph+ ALL) in the absence of BCR-ABL1. Tyrosine kinase–a