Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Laura Stahn"'
Autor:
Laura Stahn, Justyna Rasińska, Tilo Dehne, Stefanie Schreyer, Aileen Hakus, Manfred Gossen, Barbara Steiner, Shabnam Hemmati-Sadeghi
Publikováno v:
Stahn, L.; Rasińska, J.; Dehne, T.; Schreyer, S.; Hakus, A.; Gossen, M.; Steiner, B.; Hemmati-Sadeghi, S.: Sleeping beauty transposon system for GDNF overexpression of entrapped stem cells in fibrin hydrogel in a rat model of Parkinson’s disease. In: Drug Delivery and Translational Research. Vol. 13 (2023) 1745-1765. (DOI: /10.1007/s13346-023-01289-9)
There is currently no causal treatment available for Parkinson’s disease (PD). However, the use of glial cell line–derived neurotrophic factor (GDNF) to provide regenerative effects for neurons is promising. Such approaches require translational
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8db912b583701e252dda57a53fa36115
https://publications.hereon.de/id/51317
https://publications.hereon.de/id/51317
Autor:
Justyna Rasińska, Charlotte Klein, Laura Stahn, Felix Maidhof, Anna Pfeffer, Stefanie Schreyer, Manfred Gossen, Andreas Kurtz, Barbara Steiner, Shabnam Hemmati‐Sadeghi
Publikováno v:
Rasińska, J.; Klein, C.; Stahn, L.; Maidhof, F.; Pfeffer, A.; Schreyer, S.; Gossen, M.; Kurtz, A.; Steiner, B.; Hemmati-Sadeghi, S.: Transposon-mediated glial cell line-derived neurotrophic factor overexpression in human adipose tissue-derived mesenchymal stromal cells: A potential approach for neuroregenerative medicine?. In: Journal of Tissue Engineering and Regenerative Medicine. Vol. 16 (2022) 6, 515-529. (DOI: /10.1002/term.3296)
Glial cell line-derived neurotrophic factor (GDNF) has neuroprotective effects and may be a promising candidate for regenerative strategies focusing on neurodegenerative diseases. As GDNF cannot cross the blood–brain barrier to potentially regenera
Autor:
Stefanie Schreyer, Charlotte Klein, Anna Pfeffer, Justyna Rasińska, Laura Stahn, Karlotta Knuth, Basim Abuelnor, Alina Elisabeth Catharina Panzel, André Rex, Stefan Koch, Shabnam Hemmati-Sadeghi, Barbara Steiner
Publikováno v:
Scientific Reports
Scientific Reports, Vol 10, Iss 1, Pp 1-16 (2020)
Scientific Reports, Vol 10, Iss 1, Pp 1-16 (2020)
Glucose hypometabolism potentially contributes to Alzheimer’s disease (AD) and might even represent an underlying mechanism. Here, we investigate the relationship of diet-induced metabolic stress and AD as well as the therapeutic potential of chia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3bb9f5b748f3b8318acde04ae3839617
https://doi.org/10.1038/s41598-020-75209-z
https://doi.org/10.1038/s41598-020-75209-z
Publikováno v:
Aging. 8:2777-2789
Caloric restriction (CR) decelerates the aging process, extends lifespan and exerts neuroprotective effects in diverse species by so far unknown mechanisms. Based on known neuroprotective effects of fibroblastic growth factor 21 (Fgf21) we speculate
Autor:
Claire, Rühlmann, Tjark, Wölk, Tobias, Blümel, Laura, Stahn, Brigitte, Vollmar, Angela, Kuhla
Publikováno v:
Aging (Albany NY)
Caloric restriction (CR) decelerates the aging process, extends lifespan and exerts neuroprotective effects in diverse species by so far unknown mechanisms. Based on known neuroprotective effects of fibroblastic growth factor 21 (Fgf21) we speculate