Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Laura M. Montero"'
Autor:
Roberto Di Maio, Pier G. Mastroberardino, Xiaoping Hu, Laura M. Montero, J. Timothy Greenamyre
Publikováno v:
Neurobiology of Disease, Vol 49, Iss , Pp 87-98 (2013)
Hippocampal sclerosis, the main pathological sign of chronic temporal lobe epilepsy (TLE), is associated with oxidative injury, altered N-methyl d-aspartate receptor (NMDAR) stoichiometry, and loss of hippocampal neurons. However, the mechanisms that
Externí odkaz:
https://doaj.org/article/2db6d050b24c4b0da3cd9d3291ef7586
Publikováno v:
Experimental Neurology. 228:41-52
Gene transfer to the central nervous system provides powerful methodology for the study of gene function and gene-environment interactions in vivo, in addition to a vehicle for the delivery of therapeutic transgenes for gene therapy. The aim of the p
Autor:
Sara Sepe, Laura M. Montero, Xiaoping Hu, Laurie H. Sanders, J.T. Greenamyre, Roberto Di Maio, Pier G. Mastroberardino, Wiggert A. van Cappellen, Maxx P. Horowitz, Victor Tapias, Chiara Milanese, Edward A. Burton
Publikováno v:
Antioxidants & Redox Signaling, 15(4), 855-871. Mary Ann Liebert Inc.
Aims: The study of the intracellular oxido-reductive (redox) state is of extreme relevance to the dopamine (DA) neurons of the substantia nigra pars compacta. These cells possess a distinct physiology intrinsically associated with elevated reactive o
Publikováno v:
Neurobiology of Disease, Vol 36, Iss 1, Pp 96-102 (2009)
Gastrointestinal disorders, particularly severe constipation and delayed gastric emptying, are core symptoms of Parkinson's disease that affect most patients. However, the neuropathological substrate and physiological basis for this dysfunction are p
Autor:
Laura M. Montero, Roberto Di Maio, J. Timothy Greenamyre, Pier G. Mastroberardino, Xiaoping Hu
Publikováno v:
Neurobiology of Disease, Vol 49, Iss, Pp 87-98 (2013)
Neurobiology of Disease, 49, 87-98. Academic Press
Neurobiology of Disease, 49, 87-98. Academic Press
Hippocampal sclerosis, the main pathological sign of chronic temporal lobe epilepsy (TLE), is associated with oxidative injury, altered N-methyl D-aspartate receptor (NMDAR) stoichiometry, and loss of hippocampal neurons. However, the mechanisms that
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::618704fc0093b585413de2cdba3ea475
http://hdl.handle.net/11697/153586
http://hdl.handle.net/11697/153586
Autor:
Roberto Di Maio, Laura M. Montero, Xiaoping Hu, J. Timothy Greenamyre, Pier G. Mastroberardino
Publikováno v:
Neurobiology of Disease, Vol 42, Iss 3, Pp 482-495 (2011)
Neurobiology of Disease, 42(3), 482-495. Academic Press
Neurobiology of Disease, 42(3), 482-495. Academic Press
The molecular basis for epileptogenesis remains poorly defined, but repeated or prolonged seizures can cause altered hippocampal N-methyl D-aspartate receptor (NMDAR) stoichiometry, loss of hippocampal neurons, and aberrant mossy fiber sprouting. Usi