Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Laura Eve Strahle"'
Publikováno v:
Virology, Vol. 351, No 1 (2006) pp. 101-11
The ability of some Sendai virus stocks to strongly activate IFNbeta has long been known to be associated with defective-interfering (DI) genomes. We have compared SeV stocks containing various copyback and internal deletion DI genomes (and those con
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::43905e6c8d969ef9f853c41d1170c58b
https://doi.org/10.1016/j.virol.2006.03.022
https://doi.org/10.1016/j.virol.2006.03.022
Autor:
Joerg F. Schlaak, Dominique Garcin, Daniel Kolakofsky, Laura Eve Strahle, Philippe Le Mercier
Publikováno v:
Journal of Virology, Vol. 77, No 14 (2003) pp. 7903-13
We have used cDNA arrays to compare the activation of various cellular genes in response to infection with Sendai viruses (SeV) that contain specific mutations. Three groups of cellular genes activated by mutant SeV infection, but not by wild-type Se
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::84449209ea5eb56a11addb9308f7e8b2
https://doi.org/10.1128/jvi.77.14.7903-7913.2003
https://doi.org/10.1128/jvi.77.14.7903-7913.2003
Autor:
Jean-Baptiste Marq, Daniel Kolakofsky, Laura Eve Strahle, Stéphane Hausmann, Dominique Garcin, Albert Brini
Publikováno v:
Journal of Virology, Vol. 81, No 22 (2007) pp. 12227-37
As infection with wild-type (wt) Sendai virus (SeV) normally activates beta interferon (IFN-β) very poorly, two unnatural SeV infections were used to study virus-induced IFN-β activation in mouse embryonic fibroblasts: (i) SeV-DI-H4, which is compo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::65fb5aba7f6f88242d3a560a6939bd6c
https://archive-ouverte.unige.ch/unige:38151
https://archive-ouverte.unige.ch/unige:38151
Autor:
Stéphane Hausmann, Philippe Le Mercier, Daniel Kolakofsky, Laura Eve Strahle, Dominique Garcin, Philippe Plattet
Publikováno v:
Virology, Vol. 362, No 2 (2007) pp. 411-20
Mini-genomes expressing two reporter genes and a variable gene junction were used to study Sendai virus RNA polymerase (RdRp) scanning for the mRNA start signal of the downstream gene (gs2). We found that RdRp could scan the template efficiently as l
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::44b1620e2caae857802cf366a5f74795
https://pubmed.ncbi.nlm.nih.gov/17300823
https://pubmed.ncbi.nlm.nih.gov/17300823
Autor:
Philippe Le Mercier, Dominique Garcin, Jean-Baptiste Marq, Laura Eve Strahle, Daniel Kolakofsky
Publikováno v:
Virology, Vol. 295, No 2 (2002) pp. 256-65
Sendai virus infection strongly induces interferon (IFN) production and has recently been shown to interdict the subsequent IFN signaling through the Jak/Stat pathway. This anti-IFN activity of SeV is due to its “C” proteins, a nested set of four
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::58e6198c8d98e2e7b9848a3a696ca4ec