Zobrazeno 1 - 10 of 80 pro vyhledávání: '"Larry R. Boots"'
1
Androgen Excess in Women: Experience with Over 1000 Consecutive Patients
Autor: Eric S. Knochenhauer, Kent D. Taylor, K. C. Stephens, J. Lazenby, Larry R. Boots, Ricardo Azziz, Luis Sánchez, Carlos Morán
Publikováno v: The Journal of Clinical Endocrinology & Metabolism. 89:453-462
The objective of the present study was to estimate the prevalence of the different pathological conditions causing clinically evident androgen excess and to document the degree of long-term success of suppressive and/or antiandrogen hormonal therapy
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c57b6ddbf1f56ec031dd99a070a71db6
https://doi.org/10.1210/jc.2003-031122
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2
Adrenal progestogen and androgen production in 21-hydroxylase-deficient nonclassic adrenal hyperplasia is partially independent of adrenocorticotropic hormone stimulation
Autor: Luis A. Sanchez, Carlos Moran, Tatiana Ochoa, Ricardo Azziz, Larry R. Boots, Rosario Reyna
Publikováno v: Fertility and Sterility. 77:750-753
Objective: To test the hypothesis that adrenal steroidogenesis in nonclassic adrenal hyperplasia (NCAH) patients is, at least in part, independent of adrenocorticotropic hormone (ACTH) control. Design: Prospective controlled clinical study. Setting:
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e640c92042f465b98dad9a067ee8d6cd
https://doi.org/10.1016/s0015-0282(01)03236-8
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4
Adrenocortical Secretion of Dehydroepiandrosterone in Healthy Women: Highly Variable Response to Adrenocorticotropin
Autor: Ricardo Azziz, Larry R. Boots, C. Richard Parker, Liesl M. Fox, Howard A. Zacur
Publikováno v: Journal of Clinical Endocrinology & Metabolism. 86:2513-2517
Excess adrenal androgen (AA) levels are observed in 25--50% of women with the polycystic ovary syndrome (PCOS), and AA excess in PCOS may represent selection bias. Thus, it is possible that AA secretion among the general population is highly variable
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::b6f146cd3d651bcba3e7b346f55bccaf
https://doi.org/10.1210/jc.86.6.2513
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5
Adrenocortical hyperresponsivity to adrenocorticotropic hormone: a mechanism favoring the normal production of cortisol in 21-hydroxylase-deficient nonclassic adrenal hyperplasia
Autor: Larry R. Boots, Raquel Huerta, Eric S. Knochenhauer, Christine Decanter, Ricardo Azziz, Didier Dewailly
Publikováno v: Europe PubMed Central
Objective: To test the hypothesis that patients with nonclassic adrenal hyperplasia (NCAH) exhibit a generalized exaggeration in their response to ACTH stimulation that favors the normal production of F. Patients with 21-hydroxylase (21-OH)-deficient
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1c818a83756e809dd91397a81423a33a
https://doi.org/10.1016/s0015-0282(00)00631-2
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6
Effects of Aging on Adrenal Function in the Human: Responsiveness and Sensitivity of Adrenal Androgens and Cortisol to Adrenocorticotropin in Premenopausal and Postmenopausal Women1
Autor: Sejong Bae, S.M. Slayden, Ricardo Azziz, S. Lolita Crabbe, C. Richard Parker, Gene A. Hines, Larry R. Boots
Publikováno v: The Journal of Clinical Endocrinology & Metabolism. 85:48-54
We sought to determine the effects of aging on several aspects of adrenal steroidogenesis in the hopes of characterizing the possible causes of adrenal androgen deficiency in elderly women. To this end, we quantified basal morning concentrations of c
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::d30fe39f889e1ec2d25680475b15717a
https://doi.org/10.1210/jcem.85.1.6265
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7
11β-hydroxyandrostenedione and δ5-androstenediol as markers of adrenal androgen production in patients with 21-hydroxylase–deficient nonclassic adrenal hyperplasia
Autor: Christine Decanter, Larry R. Boots, Ricardo Azziz, Didier Dewailly, Eric S. Knochenhauer, Raquel Huerta
Publikováno v: Europe PubMed Central
Objective: To determine the sensitivity of 11β-hydroxyandrostenedione (11-OHA4) and Δ 5 -androstenediol (ADIOL) as markers of excessive adrenal androgen production. Design: Prospective study. Setting: Academic medical centers. Patient(s): Thirteen
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0c51d9a32476d7b17a2a125fb181e01c
https://doi.org/10.1016/s0015-0282(99)00402-1
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8
Ovulation After Glucocorticoid Suppression of Adrenal Androgens in the Polycystic Ovary Syndrome Is Not Predicted by the Basal Dehydroepiandrosterone Sulfate Level1
Autor: V. Y. Black, Eric S. Knochenhauer, Gene A. Hines, Larry R. Boots, Ricardo Azziz
Publikováno v: The Journal of Clinical Endocrinology & Metabolism. 84:946-950
Adrenal androgen (AA) excess, primarily in the form of dehydroepiandrosterone sulfate (DHEAS), affects over 50% of women with the polycystic ovary syndrome (PCOS). Nonetheless, it is unclear what role AA excess plays in the PCOS-associated oligo-ovul
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::172a057458c8337a7319c36a9394e9c8
https://doi.org/10.1210/jcem.84.3.5574
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9
Maternal serum α-fetoprotein and dimeric inhibin A detect aneuploidies other than Down syndrome
Autor: John Owen, Katharine D. Wenstrom, D.C. Chu, Larry R. Boots
Publikováno v: American Journal of Obstetrics and Gynecology. 179:966-970
Objective: Our purpose was to determine whether the combination of maternal serum α-fetoprotein, free human chorionic gonadotropin-β, dimeric inhibin A, and maternal age detects aneuploidies other than Down syndrome. Study Design: We retrieved stor
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2cb0cb29ffbddaa3628482394845a332
https://doi.org/10.1016/s0002-9378(98)70198-x
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10
Androgen Response to Hypothalamic-Pituitary-Adrenal Stimulation with Naloxone in Women with Myotonic Muscular Dystrophy
Autor: Richard V. Jackson, Liesl M. Fox, G. I. Hockings, R. P. Buyalos, G. I. Grice, David J. Torpy, Ricardo Azziz, Larry R. Boots
Publikováno v: The Journal of Clinical Endocrinology & Metabolism. 83:3219-3224
Myotonic muscular dystrophy (MMD) is a disease of autosomal dominant inheritance characterized by multisystem disease, including myotonia, muscle-wasting and weakness of all muscular tissues, and endocrine abnormalities attributed to a genetic abnorm
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::696269c4e0e2b42c7e48dd2d1411ff6f
https://doi.org/10.1210/jcem.83.9.5078
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