Re: Lanvers C et al. Pharmacology of all-trans-retinoic acid in children with acute promyelocytic leukemia. Med Pediatr Oncol 2003;40:293-301.

Autor: Formelli F

Publikováno v: Pediatric blood & cancer [Pediatr Blood Cancer] 2004 Apr; Vol. 42 (4), pp. 392-3.

Drug Monitoring of PEG-Asparaginase Treatment in Childhood Acute Lymphoblastic Leukemia and Non-Hodgkin's Lymphoma.

Autor: Pinheiro, J.P. Vieira, Lanvers, C., Wurthwein, G., Beier, R., Da Palma, J. Casimiro, Stackelberg, A. Von, Boos, J.

Publikováno v: Leukemia & Lymphoma. Oct2002, Vol. 43 Issue 10, p1911. 10p.

All-trans-retinoic acid increases cytosine arabinoside cytotoxicity in HL-60 human leukemia cells in spite of decreased cellular ara-CTP accumulation

Autor: Freund, A., Rössig, C., Lanvers, C., Gescher, A., Hohenlöchter, B., Jürgens, H., Boos, J. ^*

Publikováno v: In Annals of Oncology March 1999 10(3):335-338

Getting Away with It (Or Not): The Social Control of Organizational Deviance.

Autor: Piazza, Alessandro¹ (AUTHOR) alessandro.piazza@rice.edu, Bergemann, Patrick² (AUTHOR) pbergema@uci.edu, Helms, Wesley³ (AUTHOR) whelms@brocku.ca

Publikováno v: Academy of Management Review. Apr2024, Vol. 49 Issue 2, p249-272. 24p.

Asparagine synthetase activity in paediatric acute leukaemias:AML-M5 subtype shows lowest activity

Autor: Dübbers, A., Würthwein, G., Müller, H. J., Schulze-Westhoff, P., Winkelhorst, M., Kurzknabe, E., Lanvers, C., Pieters, R., Kaspers, G. J.L., Creutzig, U., Ritter, J., Boos, J.

Publikováno v: Dübbers, A, Würthwein, G, Müller, H J, Schulze-Westhoff, P, Winkelhorst, M, Kurzknabe, E, Lanvers, C, Pieters, R, Kaspers, G J L, Creutzig, U, Ritter, J & Boos, J 2000, ' Asparagine synthetase activity in paediatric acute leukaemias : AML-M5 subtype shows lowest activity ', British Journal of Haematology, vol. 109, no. 2, pp. 427-429 . https://doi.org/10.1046/j.1365-2141.2000.02015.x
British Journal of Haematology, 109(2), 427-429. Wiley-Blackwell

Lack of sufficient cellular activity of asparagine synthetase (AS) in blast cells compared with normal tissue is thought to be the basis of the antileukaemic effect of L-asparaginase in acute lymphoblastic leukaemia (ALL). Although L-asparaginase is

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