Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Landi V. C. Guillermo"'
Autor:
Emmanuele D. S. Andrade, Ronimara A. Santos, Landi V. C. Guillermo, Noriyuki Miyoshi, Danielly C. Ferraz da Costa
Publikováno v:
Molecules, Vol 29, Iss 19, p 4575 (2024)
Green tea is the second most consumed beverage following water, and the health benefits provided by its consumption have been well established from research in recent decades. The main bioactive compounds found in all Camellia sinensis-based teas are
Externí odkaz:
https://doaj.org/article/91a9f6ea85df45d09d688e7d5f31cb15
Autor:
Wânia F. Pereira-Manfro, Alessandra A. Filardy, Natália S Vellozo, Elisabeth M. Silva, Richard M. Siegel, Landi V. C. Guillermo, Marcela F. Lopes, George A. DosReis, Flávia L. Ribeiro-Gomes
Publikováno v:
Journal of Leukocyte Biology. 95:347-355
We investigated how apoptosis pathways mediated by death receptors and caspase-8 affect cytokine responses and immunity to Leishmania major parasites. Splenic CD4 T cells undergo activation-induced apoptosis, and blockade of FasL-Fas interaction incr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2251680348ea6f5d565f9aa989d53ddf
https://doi.org/10.1189/jlb.0912463
https://doi.org/10.1189/jlb.0912463
Autor:
Landi V. C. Guillermo, Sandro Gomes Soares, Renato Augusto DaMatta, Marcos Fernando de Rezende Matta, Marco Cesar Cunegundes Guimarães
Publikováno v:
Veterinary Immunology and Immunopathology. 140:317-322
Macrophages are fundamental cells of the innate immune system, which, through phagocytosis and nitric oxide production, eliminate pathogens. The aim of the present study was to determine if macrophages from chicken families divergently selected to hi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1acd3459f3b79520411edc7a036f6efc
https://doi.org/10.1016/j.vetimm.2011.01.004
https://doi.org/10.1016/j.vetimm.2011.01.004
Autor:
Marise P. Nunes, Juliana F. V. Senra, Landi V. C. Guillermo, Juliana de Meis, Flávia L. Ribeiro-Gomes, George A. DosReis, Elisabeth M. Silva, Milena Botelho Pereira Soares, Marcela F. Lopes
Publikováno v:
European Journal of Immunology. 37:738-746
In experimental Chagas' disease, lymphocytes from mice infected with Trypanosoma cruzi show increased apoptosis in vivo and in vitro. Treatment with a pan-caspase blocker peptide inhibited expression of the active form of effector caspase-3 in vitro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5e02512063a1483c5b35515296f0f7d5
https://doi.org/10.1002/eji.200636790
https://doi.org/10.1002/eji.200636790
Publikováno v:
Cytokine & Growth Factor Reviews. 18:97-105
Parasitic diseases have worldwide medical and economical impact. Host T lymphocytes and the cytokines they produce determine the outcome of parasitic infections. Programmed cell death by apoptosis is induced in the course of parasitic infections, and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e5afba9734572c3f1c933a531dfb9cc6
https://doi.org/10.1016/j.cytogfr.2007.01.009
https://doi.org/10.1016/j.cytogfr.2007.01.009
Autor:
Marcela F. Lopes, Flávia L. Ribeiro-Gomes, Hideo Yagita, Elisabeth M. Silva, Juliana de Meis, Landi V. C. Guillermo, George A. DosReis, Wânia F. Pereira
Publikováno v:
Journal of Leukocyte Biology. 81:942-951
We investigated the role of the Fas ligand (FasL)/Fas death pathway on apoptosis and cytokine production by T cells in Trypanosoma cruzi infection. Anti-FasL, but not anti-TNF-α or anti-TRAIL, blocked activation-induced cell death of CD8 T cells and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9320b59572172ada8e231882e27b8292
https://doi.org/10.1189/jlb.1006643
https://doi.org/10.1189/jlb.1006643
Autor:
Landi V. C. Guillermo, Renata M. Pereira, Richard M. Siegel, Marcela F. Lopes, Sérgio H. Seabra, Elisabeth M. Silva, Flávia L. Ribeiro-Gomes, Ulisses Gazos Lopes, Teresa Cristina Calegari-Silva, Zhengqi Wu, George A. DosReis, Juliana de Meis
Publikováno v:
The Journal of Immunology. 174:6314-6321
During Trypanosoma cruzi infection, T cells up-regulate caspase-8 activity. To assess the role of caspase-8 in T cell-mediated immunity, we investigated the effects of caspase-8 inhibition on T cells in viral FLIP (v-FLIP) transgenic mice. Compared w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::434af42c5dab59fbd1f830e14110bdb8
https://doi.org/10.4049/jimmunol.174.10.6314
https://doi.org/10.4049/jimmunol.174.10.6314
Autor:
Andrade, Emmanuele D. S.1,2 (AUTHOR) manu.dutra.pbi@gmail.com, Santos, Ronimara A.2 (AUTHOR), Guillermo, Landi V. C.3 (AUTHOR) landiguillermo@gmail.com, Miyoshi, Noriyuki1 (AUTHOR) miyoshin@u-shizuoka-ken.ac.jp, Ferraz da Costa, Danielly C.2 (AUTHOR) miyoshin@u-shizuoka-ken.ac.jp
Publikováno v:
Molecules. Oct2024, Vol. 29 Issue 19, p4575. 15p.
Autor:
Christina Maeda Takiya, Landi V. C. Guillermo, Flávia L. Ribeiro-Gomes, Elisabeth M. Silva, Juliana de Meis, Marcela F. Lopes, Karina Kroll-Palhares, Wânia F. Pereira
Publikováno v:
Immunopharmacology and immunotoxicology. 31(2)
Caspases are cysteine aspartases acting either as initiators (caspases 8, 9, and 10) or executioners (caspases 3, 6, and 7) to induce programmed cell death by apoptosis. Parasite infections by certain intracellular protozoans increase host cell life
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8749aa70ae8c20aa32349fa3a5803b2b
https://pubmed.ncbi.nlm.nih.gov/18785049
https://pubmed.ncbi.nlm.nih.gov/18785049
Autor:
George A. DosReis, Elisabeth M. Silva, Marcela F. Lopes, Juliana de Meis, Landi V. C. Guillermo, Lidia M. S. Ferreira
Publikováno v:
European journal of immunology. 38(1)
Infection with Trypanosoma cruzi causes expansion of subcutaneous (SLN) and atrophy of mesenteric (MLN) lymph nodes. Here we show that excision of MLN increased parasitemia in T. cruzi-infected mice. We then studied how apoptosis of MLN cells affects
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b9f749a6571cfa35e9f124693e72f42c
https://pubmed.ncbi.nlm.nih.gov/18085669
https://pubmed.ncbi.nlm.nih.gov/18085669