Zobrazeno 1 - 10
of 41
pro vyhledávání: '"Laird Miers"'
Publikováno v:
PLoS ONE, Vol 13, Iss 9, p e0204536 (2018)
[This corrects the article DOI: 10.1371/journal.pone.0167573.].
Externí odkaz:
https://doaj.org/article/a0f28c54c2944e759631a31fd0f5c776
Publikováno v:
PLoS ONE, Vol 11, Iss 12, p e0167573 (2016)
Charcot-Marie-Tooth disease type 2A (CMT2A), the most common axonal form of hereditary sensory motor neuropathy, is caused by mutations of mitofusin-2 (MFN2). Mitofusin-2 is a GTPase required for fusion of mitochondrial outer membranes, repair of dam
Externí odkaz:
https://doaj.org/article/164a7964088747d0b17872301df0558a
Autor:
Jiho Sohn, Peter Bannerman, Fuzheng Guo, Travis Burns, Laird Miers, Christopher Croteau, Naveen K. Singhal, Jennifer A. McDonough, David Pleasure
Publikováno v:
The Journal of Neuroscience. 37:413-421
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c9f5d5749aa5b1044c2a65bccb5f984b
https://doi.org/10.1523/jneurosci.2013-16.2017
https://doi.org/10.1523/jneurosci.2013-16.2017
Publikováno v:
Journal of Neuroimmunology. 290:36-46
Studies in multiple sclerosis and its animal model experimental autoimmune encephalomyelitis (EAE) suggest that peripheral monocyte-derived cells (MDCs) are instrumental for disease initiation. MDCs, however, are plastic, and may exert various functi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5ec6519b36755b49aba82d4eca0cddb7
https://doi.org/10.1016/j.jneuroim.2015.11.004
https://doi.org/10.1016/j.jneuroim.2015.11.004
Autor:
Jie Xu, Laird Miers, Fuzheng Guo, Shuo Li, Travis Burns, Peter Bannerman, Emily Mills Ko, David E Pleasure, Jennifer McDonough, Ernest J. Freeman
Publikováno v:
Annals of Neurology. 77:884-888
Canavan disease is caused by inactivating ASPA (aspartoacylase) mutations that prevent cleavage of N-acetyl-L-aspartate (NAA), resulting in marked elevations in central nervous system (CNS) NAA and progressively worsening leukodystrophy. We now repor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ad90c17a669d33a3a1918779f8095bc9
https://doi.org/10.1002/ana.24392
https://doi.org/10.1002/ana.24392
Autor:
Fuzheng Guo, Peter Bannerman, Joyce Ma, Athena M. Soulika, Monica Moreno, Laird Miers, David E Pleasure
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience, vol 34, iss 24
Current multiple sclerosis (MS) therapies only partially prevent chronically worsening neurological deficits, which are largely attributable to progressive loss of CNS axons. Prior studies of experimental autoimmune encephalomyelitis (EAE) induced in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dc42bb1320979eef80330eb8d4efd9c3
https://doi.org/10.1523/jneurosci.1137-14.2014
https://doi.org/10.1523/jneurosci.1137-14.2014
Publikováno v:
PLoS ONE
PLoS ONE, Vol 11, Iss 12, p e0167573 (2016)
PloS one, vol 11, iss 12
PLoS ONE, Vol 11, Iss 12, p e0167573 (2016)
PloS one, vol 11, iss 12
Charcot-Marie-Tooth disease type 2A (CMT2A), the most common axonal form of hereditary sensory motor neuropathy, is caused by mutations of mitofusin-2 (MFN2). Mitofusin-2 is a GTPase required for fusion of mitochondrial outer membranes, repair of dam
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::288bf2477335b9fcede271170f9ac528
https://pubmed.ncbi.nlm.nih.gov/30226905
https://pubmed.ncbi.nlm.nih.gov/30226905
Autor:
Peter Bannerman, Joyce Ma, Laird Miers, Fuzheng Guo, Chengji J. Zhou, Emily Mills Ko, Yazhou Wang, Jiho Sohn, Jie Xu, Hirohide Takebayashi, David E Pleasure, Monica Delgado, Yoshiko Maeda
Publikováno v:
The Journal of Neuroscience. 31:11914-11928
Accumulations of hypertrophic, intensely glial fibrillary acidic protein-positive (GFAP+) astroglia, which also express immunoreactive nestin and vimentin, are prominent features of multiple sclerosis lesions. The issues of the cellular origin of hyp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a387bcd0955b0b1e1b04fa3418e740e4
https://doi.org/10.1523/jneurosci.1759-11.2011
https://doi.org/10.1523/jneurosci.1759-11.2011
Autor:
Flora M. Vaccarino, Laird Miers, Jie Xu, Fuzheng Guo, David E Pleasure, Yoshiko Maeda, Joyce Ma, Makoto Horiuchi
Publikováno v:
The Journal of Neuroscience. 30:12036-12049
Previous studies have shown that oligodendroglial progenitor cells (OPCs) can give rise to neuronsin vitroand in perinatal cerebral cortexin vivo. We now report that OPCs in adult murine piriform cortex express low levels of doublecortin, a marker fo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::15ea59d69025c6d4e8489152aacefc0a
https://doi.org/10.1523/jneurosci.1360-10.2010
https://doi.org/10.1523/jneurosci.1360-10.2010
Autor:
Fuzheng, Guo, Peter, Bannerman, Emily, Mills Ko, Laird, Miers, Jie, Xu, Travis, Burns, Shuo, Li, Ernest, Freeman, Jennifer A, McDonough, David, Pleasure
Publikováno v:
Annals of neurology. 77(5)
Canavan disease is caused by inactivating ASPA (aspartoacylase) mutations that prevent cleavage of N-acetyl-L-aspartate (NAA), resulting in marked elevations in central nervous system (CNS) NAA and progressively worsening leukodystrophy. We now repor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::f362f790939cba0965b587687443dbed
https://pubmed.ncbi.nlm.nih.gov/25712859
https://pubmed.ncbi.nlm.nih.gov/25712859