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Cellular senescence drives age-dependent hepatic steatosis

Autor: Ogrodnik, Mikolaj, Miwa, Satomi, Tchkonia, Tamar, Tiniakos, Dina, Wilson, Caroline L., Lahat, Albert, Day, Christoper P., Burt, Alastair, Palmer, Allyson, Anstee, Quentin M., Grellscheid, Sushma Nagaraja, Hoeijmakers, Jan H J., Barnhoorn, Sander, Mann, Derek A., Bird, Thomas G., Vermeij, Wilbert P., Kirkland, James L., Passos, João F., von Zglinicki, Thomas, Jurk, Diana

Publikováno v: Nature Communications, Vol 8, Iss 1, Pp 1-12 (2017)
Ogrodnik, M, Miwa, S, Tchkonia, T, Tiniakos, D, Wilson, C L, Lahat, A, Day, C P, Burt, A, Palmer, A, Anstee, Q M, Grellscheid, S N, Hoeijmakers, J H J, Barnhoorn, S, Mann, D A, Bird, T G, Vermeij, W P, Kirkland, J L, Passos, J F, Von Zglinicki, T & Jurk, D 2017, ' Cellular senescence drives age-dependent hepatic steatosis ', Nature Communications, vol. 8, pp. 15691 . https://doi.org/10.1038/ncomms15691
Nature Communications, 8:15691. Nature Publishing Group
Nature communications, 2017, Vol.8, pp.15691 [Peer Reviewed Journal]

The incidence of non-alcoholic fatty liver disease (NAFLD) increases with age. Cellular senescence refers to a state of irreversible cell-cycle arrest combined with the secretion of proinflammatory cytokines and mitochondrial dysfunction. Senescent c

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::0f25e54194303d19b954906831379f95
https://doaj.org/article/59da7366bd92469288a45b90a4b396f9

Differential mRNA Alternative Splicing.

Autor: Lahat, Albert, Grellscheid, Sushma Nagaraja

Publikováno v: Field Guidelines for Genetic Experimental Designs in High-Throughput Sequencing; 2016, p105-119, 15p