Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Lachlan, Baer"'
Autor:
Lachlan Baer, Karissa Barthelson, John H Postlethwait, David L Adelson, Stephen M Pederson, Michael Lardelli
Publikováno v:
PLoS Computational Biology, Vol 20, Iss 2, p e1011868 (2024)
In comparisons between mutant and wild-type genotypes, transcriptome analysis can reveal the direct impacts of a mutation, together with the homeostatic responses of the biological system. Recent studies have highlighted that, when the effects of hom
Externí odkaz:
https://doaj.org/article/d9c21ff053ad40258f64fe602bea9ede
Publikováno v:
BMC Research Notes, Vol 14, Iss 1, Pp 1-5 (2021)
Abstract Objective NGFR/p75NTR and NRADD/NRH proteins are closely related structurally and are encoded by genes that arose from a duplication event early in vertebrate evolution. The transmembrane domain (TMD) of NGFR is cleaved by γ-secretase but t
Externí odkaz:
https://doaj.org/article/ac89740387da41a380f6ce0fb9f9d4a3
Autor:
Karissa Barthelson, Lachlan Baer, Yang Dong, Melanie Hand, Zac Pujic, Morgan Newman, Geoffrey J. Goodhill, Robert I. Richards, Stephen M. Pederson, Michael Lardelli
Publikováno v:
Frontiers in Genetics, Vol 12 (2021)
Zebrafish represent a valuable model for investigating the molecular and cellular basis of Fragile X syndrome (FXS). Reduced expression of the zebrafish FMR1 orthologous gene, fmr1, causes developmental and behavioural phenotypes related to FXS. Zebr
Externí odkaz:
https://doaj.org/article/8c74c3c0e3554ed1a8851000275ae0a5
Autor:
Lachlan Baer, Karissa Barthelson, John Postlethwait, David Adelson, Stephen Pederson, Michael Lardelli
Publikováno v:
bioRxiv
In comparisons between mutant and wild-type genotypes, transcriptome analysis can reveal the direct impacts of a mutation, together with the homeostatic responses of the biological system. Recent studies have highlighted that, when homozygous mutatio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0c4d28b7c2ecf7e6967b413c6737e851
https://europepmc.org/articles/PMC10028786/
https://europepmc.org/articles/PMC10028786/
Autor:
Karissa, Barthelson, Lachlan, Baer, Yang, Dong, Melanie, Hand, Zac, Pujic, Morgan, Newman, Geoffrey J, Goodhill, Robert I, Richards, Stephen M, Pederson, Michael, Lardelli
Publikováno v:
Frontiers in Genetics
Zebrafish represent a valuable model for investigating the molecular and cellular basis of Fragile X syndrome (FXS). Reduced expression of the zebrafish FMR1 orthologous gene, fmr1, causes developmental and behavioural phenotypes related to FXS. Zebr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::5f44c504aa4443976804a92c7260df47
https://pubmed.ncbi.nlm.nih.gov/34135935
https://pubmed.ncbi.nlm.nih.gov/34135935