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Autor:
J. A. F. Demandt, L. J. Dubois, K. van Kuijk, M. Zaťovičová, H. Jin, S. Parkkila, S. W. van der Laan, L. Jelenska, B. M. E. Mees, C. P. M. Reutelingsperger, K. B. J. M. Cleutjens, C. J. H. van der Kallen, C. G. Schalkwijk, M. M. J. van Greevenbroek, E. A. L. Biessen, G. Pasterkamp, S. Pastoreková, C. D. A. Stehouwer, J. C. Sluimer
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-14 (2021)
Abstract Hypoxia is prevalent in atherosclerotic plaques, promoting plaque aggravation and subsequent cardiovascular disease (CVD). Transmembrane protein carbonic anhydrase IX (CAIX) is hypoxia-induced and can be shed into the circulation as soluble
Externí odkaz:
https://doaj.org/article/b88f767adcdb47aab623eaef420a2b57