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pro vyhledávání: '"L. Elizabeth Caldwell"'
Autor:
Katherine B. Hobbs, L. Elizabeth Caldwell, Eric C. Dietze, Kevin Heine, Randy O. Sigle, Victoria L. Seewaldt, Krzysztof Mrózek, David M. Hockenbery
Publikováno v:
The Journal of Cell Biology
Little is known about the fate of normal human mammary epithelial cells (HMECs) that lose p53 function in the context of extracellular matrix (ECM)-derived growth and polarity signals. Retrovirally mediated expression of human papillomavirus type 16
Autor:
Kanur Srinivasan, L. Elizabeth Caldwell, Eric C. Dietze, Molly B. Parker, Victoria L. Seewaldt, Ji-Hyon Kim
Publikováno v:
Experimental Cell Research. 249:70-85
Overexpression of cyclin D1 protein is observed in the majority of breast cancers, suggesting that dysregulated expression of cyclin D1 might be a critical event in breast cancer carcinogenesis. We investigated whether retroviral-mediated expression
Autor:
Michelle L. Bowie, Kazunari K. Yokoyama, Michelle M. Troch, L. Elizabeth Caldwell, Victoria L. Seewaldt, Cassandra L. Neal, Robin J. Marjoram, Krzysztof Mrózek, Gregory R. Bean, Eric C. Dietze, Catherine Ibarra
Publikováno v:
Journal of cell science. 118(Pt 21)
Interactions between normal mammary epithelial cells and extracellular matrix (ECM) are important for mammary gland homeostasis. Loss of interactions between ECM and normal mammary epithelial cells are thought to be an early event in mammary carcinog
Autor:
Katherine B. Hobbs, Lisa D. Yee, Kelly Marcom, Karen Swisshelm, Victoria L. Seewaldt, Steven J. Collins, Gregory R. Bean, Eric C. Dietze, L. Elizabeth Caldwell
Publikováno v:
Microscopy research and technique. 59(1)
Retinoids and retinoic acid receptors (RARs) are important mediators of normal epithelial cell homeostasis. To assess the role of retinoids and RARs in regulating growth arrest and apoptosis in benign and malignant mammary epithelial cells, two model
Autor:
Mariangela Mancini, Victoria L. Seewaldt, L. Elizabeth Caldwell, Svetlana L. Grupin, Eric C. Dietze
Publikováno v:
Web of Science
Despite the widespread clinical use of tamoxifen as a breast cancer prevention agent, the molecular mechanism of tamoxifen chemoprevention is poorly understood. Abnormal expression of p53 is felt to be an early event in mammary carcinogenesis. We dev
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::73966829ea10122b4214de7e6aa9eac4
https://publons.com/wos-op/publon/32607793/
https://publons.com/wos-op/publon/32607793/
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