Zobrazeno 1 - 10
of 39
pro vyhledávání: '"L. Abu-Tair"'
Akademický článek
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Autor:
S. Doron, R. Ghantous, Rifaat Safadi, Mahmud Mahamid, Johnny Amer, L. Abu-Tair, A. Salhab, Mazen Noureddin
Publikováno v:
Hepatology Communications
Insulin resistance is a key risk factor in the progression of nonalcoholic fatty liver disease (NAFLD) and may lead to liver fibrosis. Natural killer (NK) cells are thought to exert an antifibrotic effect through their killing of activated hepatic st
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::97096f55c78c01462e07c482c91d77d1
https://doi.org/10.1002/hep4.1146
https://doi.org/10.1002/hep4.1146
Autor:
Rufayda Mruwat, S. Doron, Rami Grifat, Areej El-Khatib, Johnny Amer, L. Abu-Tair, Rifaat Safadi
Publikováno v:
Liver International. 34:1232-1240
Immune cells interact with hepatic-stellate-cells (HSCs) in the development of liver fibrosis. Little is known about the influence of pregnancy on the development and progression of hepatic-fibrosis. In this study, we explored the influence of pregna
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bd3bb1a207f17202fe3dbd2bd898351c
https://doi.org/10.1111/liv.12371
https://doi.org/10.1111/liv.12371
Autor:
Areej Khatib, Yosefa Avraham, Rifaat Safadi, Elliot M. Berry, Johnny Amer, L. Abu-Tair, Mahmud Mahamid, S. Doron
Publikováno v:
American Journal of Physiology-Gastrointestinal and Liver Physiology. 302:G1364-G1372
Cannabinoid 2 (CB2) receptors expressed on immune cells are considered to be antifibrogenic. Hepatic stellate cells (HSCs) directly interact with phagocytosis lymphocytes, but the nature of this interaction is obscure. We aimed to study the effects o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::55e6761bba41716c3a5a535890876781
https://doi.org/10.1152/ajpgi.00191.2011
https://doi.org/10.1152/ajpgi.00191.2011
Akademický článek
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Autor:
S. Doron, Yossi Ovadya, Eithan Galun, Valery Krizhanovsky, Rifaat Safadi, Johnny Amer, Jonathan H. Axelrod, L. Abu-Tair
Publikováno v:
PLoS ONE
PLoS ONE, Vol 8, Iss 12, p e82571 (2013)
PLoS ONE, Vol 8, Iss 12, p e82571 (2013)
The toll-like receptor-9 (TLR9) agonist cytosine phosphate guanine (CpG), activates hepatic stellate cells (HSCs) and mediates fibrosis. We investigated the TLR9 effects on lymphocyte/HSCs interactions. Liver fibrosis was induced in wild-type (WT) mi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c78c42778a0e74d132d068bc6f4a385d
http://europepmc.org/articles/PMC3858328
http://europepmc.org/articles/PMC3858328
Autor:
Rifaat Safadi, L. Abu-Tair, Nidal Muhanna, Johnny Amer, Mahmud Mahamid, S. Doron, Hiba Zayyad
Publikováno v:
Mitochondrion. 13(5)
We studied the in-vitro / in-vivo interactions between HCC/HSCs in early and advanced fibrosis-models. Hep3B-mono-cultures secreted high levels of αFetoProtein (αFP). Human-HSCs co-cultured with Hep3B-cells significantly decreased αFP and increase
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ad93542096d20432a9ce2f4d86b4a285
https://pubmed.ncbi.nlm.nih.gov/23333797
https://pubmed.ncbi.nlm.nih.gov/23333797
Publikováno v:
Mitochondrion. 13(5)
We investigated leptin effects on lymphocyte interactions with hepatic-stellate-cells (HSCs). Leptin showed pro-fibrotic effects on HSCs with oxidative status imbalance. In co-cultures, leptin activates HSCs and consequently adhered HCV-lymphocytes m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6986013d362ad169ff38b09b1770aade
https://pubmed.ncbi.nlm.nih.gov/23092931
https://pubmed.ncbi.nlm.nih.gov/23092931
Publikováno v:
Journal of Hepatology. 54:S39-S40
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::df74c7981d5749c9d968fa822a6601e6
https://doi.org/10.1016/s0168-8278(11)60090-1
https://doi.org/10.1016/s0168-8278(11)60090-1
Publikováno v:
Journal of Hepatology. 54:S238-S239
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::db68c22746c78d3295f16ed94afcd0e6
https://doi.org/10.1016/s0168-8278(11)60588-6
https://doi.org/10.1016/s0168-8278(11)60588-6