Zobrazeno 1 - 10
of 28
pro vyhledávání: '"L D Quarles"'
Publikováno v:
American Journal of Physiology-Endocrinology and Metabolism. 295:E254-E261
Autosomal recessive hypophosphatemic rickets (ARHR), which is characterized by renal phosphate wasting, aberrant regulation of 1α-hydroxylase activity, and rickets/osteomalacia, is caused by inactivating mutations of dentin matrix protein 1 ( DMP1).
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b135643433cecf9c2bc20d41ebc8705
https://doi.org/10.1152/ajpendo.90201.2008
https://doi.org/10.1152/ajpendo.90201.2008
Autor:
Di Chen, Gloria Gutierrez, J Cundy, Y. Kumagai, L. D. Quarles, S Navvab, R Blacher, Gregory R. Mundy, Peter S. N. Rowe, I R Garrett, David M. Rosen, Marc K. Drezner
Publikováno v:
Bone. 34:303-319
Matrix extracellular phosphoglycoprotein (MEPE) is expressed exclusively in osteoblasts, osteocytes and odontoblasts with markedly elevated expression found in X-linked hypophosphatemic rickets (Hyp) osteoblasts and in oncogenic hypophosphatemic oste
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::88e8a50e49f7b9e221b61e48f79fc9fe
https://doi.org/10.1016/j.bone.2003.10.005
https://doi.org/10.1016/j.bone.2003.10.005
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 83:3860-3866
Several factors have been identified as important in the pathogenesis of secondary hyperparathyroidism in end-stage renal disease, including serum calcium, phosphorus, and calcitriol. To examine the independent effects of key factors, we prospectivel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4992975812146174682d8ab738343b2c
https://doi.org/10.1210/jcem.83.11.5234
https://doi.org/10.1210/jcem.83.11.5234
Autor:
O S Indridason, L D Quarles
Publikováno v:
Current Opinion in Nephrology and Hypertension. 4:307-312
Deficiency of 1,25-dihydroxyvitamin D plays an important role in the pathogenesis of secondary hyperparathyroidism. Adequate replacement of this hormone is required to normalize parathyroid gland function and restore bone homeostasis in patients with
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ea15b70a661bcf505c6a13f1cccb5f5f
https://doi.org/10.1097/00041552-199507000-00004
https://doi.org/10.1097/00041552-199507000-00004
Publikováno v:
American Journal of Physiology-Endocrinology and Metabolism. 264:E938-E942
Glucocorticoids induce bone loss by increasing bone resorption and decreasing bone formation. In this study we have investigated the potential of salmon calcitonin to attenuate glucocorticoid-induced bone loss in a dog model. Male beagles were divide
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::70e88c498863d7df3109ed729b434952
https://doi.org/10.1152/ajpendo.1993.264.6.e938
https://doi.org/10.1152/ajpendo.1993.264.6.e938
Autor:
L. D. Quarles
Publikováno v:
Journal of Clinical Endocrinology & Metabolism. 75:145-150
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::738f35a69a5c283651d53c0af738f877
https://doi.org/10.1210/jc.75.1.145
https://doi.org/10.1210/jc.75.1.145
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 75:145-150
To examine the possibility that uremia alters the relationship between bioactive PTH serum concentrations and its osseous end-organ response, we evaluated the relationship between circulating intact PTH and bone turnover in 39 end-stage renal disease
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c5ba00da216bdb20e48ac545c179178b
https://doi.org/10.1210/jcem.75.1.1619003
https://doi.org/10.1210/jcem.75.1.1619003
Publikováno v:
New England Journal of Medicine. 325:544-550
Osteopenia is a major complication of renal transplantation. Immunosuppressive regimens including cyclosporine, which permit the use of lower doses of glucocorticoids, may reduce glucocorticoid-induced osteopenia.We prospectively studied the magnitud
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cb93a6c93862790857096ba2a96fa054
https://doi.org/10.1056/nejm199108223250804
https://doi.org/10.1056/nejm199108223250804
Publikováno v:
American journal of physiology. Endocrinology and metabolism. 281(4)
X-linked hypophosphatemia (XLH) is caused by inactivating mutations of Phex, a phosphate-regulating endopeptidase. Further advances in our knowledge of the pathogenesis of XLH require identification of the biological function of Phex and its physiolo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d80ca90ebe46f9f22bf3c04230557e2a
https://pubmed.ncbi.nlm.nih.gov/11551862
https://pubmed.ncbi.nlm.nih.gov/11551862
Publikováno v:
The Journal of biological chemistry. 275(5)
We isolated osteoblastic cell lines from wild-type (CasR(+/+)) and receptor null (CasR(-/-)) mice to investigate whether CasR is present in osteoblasts and accounts for their responses to extracellular cations. Osteoblasts from both CasR(+/+) and Cas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::63d05be5c83b14bdcfe6493914159dd5
https://pubmed.ncbi.nlm.nih.gov/10652312
https://pubmed.ncbi.nlm.nih.gov/10652312