Zobrazeno 1 - 5
of 5
pro vyhledávání: '"L A, Bentivegna"'
Publikováno v:
Cardiovascular Research. 27:199-203
Objective: Calcium transients in muscles from patients with end stage heart failure consist of two components (L1 and L2) at physiological extracellular calcium concentrations ([Ca2+]); the second component (L2) can appear in normal human myocardium
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aa404364095b773e58c705d195ebd6c8
https://doi.org/10.1093/cvr/27.2.199
https://doi.org/10.1093/cvr/27.2.199
Publikováno v:
Journal of Clinical Investigation. 89:2060-2065
Ischemia-induced ventricular dysfunction has been shown to be associated with increased diastolic and systolic intracellular concentrations of free, ionized calcium ([Ca2+]i). The present study was designed to determine the effects of the Ca2+ antago
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::998ecfb8e1e7e845131801bb780a7878
https://doi.org/10.1172/jci115818
https://doi.org/10.1172/jci115818
Publikováno v:
Circulation Research. 69:1538-1545
The purpose of this study was to test the hypothesis that systolic and diastolic dysfunction in left ventricular pressure-overload hypertrophy is caused by abnormal intracellular calcium handling. Experiments were performed with intact, buffer-perfus
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::50a82a26d044ba438f54cdf1590dc966
https://doi.org/10.1161/01.res.69.6.1538
https://doi.org/10.1161/01.res.69.6.1538
Publikováno v:
Circulation research. 71(6)
The purpose of this study was to test the hypothesis that abnormal intracellular calcium handling characterizes myocardial stunning. Isolated, isovolumic, buffer-perfused ferret hearts were loaded with the bioluminescent calcium indicator aequorin fo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7673e2a3de6efb71af41532c433d8b5b
https://pubmed.ncbi.nlm.nih.gov/1423931
https://pubmed.ncbi.nlm.nih.gov/1423931
Publikováno v:
European heart journal. 11
Acute or chronic heart failure may be caused by one or more of a variety of abnormalities including changes in excitation-contraction coupling processes (i.e. decreased availability of activator Ca2+ or a change in myofilament Ca2+ responsiveness), a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f260d278378b012f718539ccc35986f
https://pubmed.ncbi.nlm.nih.gov/2188844
https://pubmed.ncbi.nlm.nih.gov/2188844