Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Kyu-dong Yoo"'
Autor:
Kyu-Dong Yoo, Eun-Seok Park, Yong Lim, Shin-Il Kang, Su-Hyang Yoo, Ha-Hee Won, Young-Hee Kim, Ick-Dong Yoo, Hwan-Soo Yoo, Jin Tae Hong, Yeo-Pyo Yun
Publikováno v:
Journal of Pharmacological Sciences, Vol 118, Iss 2, Pp 171-177 (2012)
Abnormal proliferation of vascular smooth muscle cells (VSMCs) plays an essential role in the pathogenesis of vascular diseases, such as atherosclerosis, hypertension, and restenosis. Clitocybin A, a novel isoindolinone, isolated from the culture bro
Externí odkaz:
https://doaj.org/article/bfffdf1ddcb14471b2ca06eab9a9af88
Autor:
Jin-Tae Hong, Yeo-Pyo Yun, Su-Hyang Yoo, Yong Lim, Kyu-Dong Yoo, Hwan-Soo Yoo, Mi-Yea Lee, Seung-Jung Kim
Publikováno v:
Vascular pharmacology. 59(1-2)
Vascular diseases such as atherosclerosis and restenosis artery angioplasty are associated with vascular smooth muscle cell (VSMC) proliferation and intimal thickening arterial walls. In the present study, we investigated the inhibitory effects of su
Autor:
Yeo-Pyo Yun, Hwa-Sup Shin, Eun-Seok Park, Bokyung Kim, Shin-Il Kang, Jin-Tae Hong, Mi-Yea Lee, Hwan-Soo Yoo, Kyu-dong Yoo
Publikováno v:
Experimental cell research. 319(7)
The abnormal proliferation of vascular smooth muscle cells (VSMCs) in arterial wall is a major cause of vascular disorders such as atherosclerosis and restenosis after angioplasty. In this study, we investigated not only the inhibitory effects of cam
Autor:
Yong Lim, Shin-Il Kang, Su-Hyang Yoo, Ick-Dong Yoo, Young-Hee Kim, Eun-Seok Park, Yeo-Pyo Yun, Hee-Pom Lee, Kyu-Dong Yoo, Hwan-Soo Yoo, Jin-Tae Hong, Ha-Hee Won
Publikováno v:
Vascular pharmacology. 56(1-2)
The increased proliferation of vascular smooth muscle cells (VSMCs) in the arterial wall is a critical pathogenic factor for vascular diseases such as atherosclerosis and restenosis after angioplasty. Clitocybin B was reported to have either a potent
Autor:
Kyu Dong Yoo, Seung Ki Lee, Byoung Duck Park, Seung-Ju Cho, Hyun Ji Jeong, Young Mi Ham, Young Tae Je
Publikováno v:
Biochemical and biophysical research communications. 361(4)
Here we demonstrate that JNK3 can phosphorylate Smac. Smac phosphorylation attenuates its ability to activate apoptosome activity in HeLa S-100 cell lysates. Addition of the X-linked inhibitor of apoptosis protein (XIAP) to the S-100 markedly suppres