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pro vyhledávání: '"Kyell Schwartz"'
Publikováno v:
Vaccines, Vol 6, Iss 3, p 57 (2018)
β-defensins are predicted to play an important role in innate immunity against bacterial infections in the airway. We previously observed that a type III-secretion product of Bordetella bronchiseptica inhibits the NF-κB-mediated induction of a β-d
Externí odkaz:
https://doaj.org/article/2d041c9e1f4e42ad8644167b127b00c2
Publikováno v:
Infection and Immunity. 79:2250-2256
Human gingival epithelial cells (GEC) produce peptides, such as β-defensins and the cathelicidin LL-37, that are both antimicrobial and that modulate the innate immune response. In myeloid and airway epithelial cells, the active form of vitamin D 3
Autor:
Kyell Schwartz, Gill Diamond, Sunghan Yim, Nicholas J. Megjugorac, Zhiwei Yin, Joshua M. Abrahams, Lisa K. Ryan, Victoria Uhlhorn, Jihong Dai, Patricia Fitzgerald-Bocarsly
Publikováno v:
Journal of Leukocyte Biology. 90:343-356
hBD comprise a family of antimicrobial peptides that plays a role in bridging the innate and adaptive immune responses to infection. The expression of hBD-2 increases upon stimulation of numerous cell types with LPS and proinflammatory cytokines. In
Publikováno v:
Vaccines
Volume 6
Issue 3
Vaccines, Vol 6, Iss 3, p 57 (2018)
Volume 6
Issue 3
Vaccines, Vol 6, Iss 3, p 57 (2018)
&beta
defensins are predicted to play an important role in innate immunity against bacterial infections in the airway. We previously observed that a type III-secretion product of Bordetella bronchiseptica inhibits the NF-&kappa
B-mediated i
defensins are predicted to play an important role in innate immunity against bacterial infections in the airway. We previously observed that a type III-secretion product of Bordetella bronchiseptica inhibits the NF-&kappa
B-mediated i
Publikováno v:
The Journal of Immunology. 190:57.1-57.1
Beta-defensins have been gaining recognition as immunomodulators and regulators of the inflammatory response. Human beta-defensin (hBD) peptides 1, 2 and 3 are differentially modulated in primary gingival and tracheal epithelial cells (EC) by viruses