Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Kunyue Xue"'
Autor:
Caili Zhuo, Jie Lan, Xue Li, Yao Wu, Ruli Li, Ling-yu Li, Xin Yan, Hongying Chen, Li Fu, Xuemei Chen, Juanjuan Xin, Kunyue Xue, Ming-ming Tong, He Li, Xiaoxiao Wang, Jiayi Xu, Yanjing Zhang, Sisi Wu, Wei Jiang
Publikováno v:
Cell Biology and Toxicology. 39:237-258
Sirt6, a class III NAD+-dependent deacetylase of the sirtuin family, is a highly specific H3 deacetylase and plays important roles in regulating cellular growth and death. The induction of oxidative stress and death is the critical mechanism involved
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e45dfb2b168df48b73370359c39097a5
https://doi.org/10.1007/s10565-021-09649-2
https://doi.org/10.1007/s10565-021-09649-2
Autor:
Ruli Li, Caili Zhuo, Kunyue Xue, He Li, Wei Jiang, Xin Yan, Juanjuan Xin, Xu-Lei Wang, Xue Li, Jie Lan, Xiao-Xiao Wang, Heng-Yu Zhang, Ling-yu Li, Zi-jie Deng, Jia-Xiang Chen, Lu Gan, Ming-ming Tong
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1865:2379-2392
Background Abnormalities of the L-arginine-nitric oxide pathway induce hypertension. 5-Lipoxygenase (5-LO) is the key enzyme involved in synthesis of leukotrienes (LTs). However, whether nitricoxide synthase dysfunction induces hypertensive vascular
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1ca45fcddbb81e4babf46d41aed837af
https://doi.org/10.1016/j.bbadis.2019.05.021
https://doi.org/10.1016/j.bbadis.2019.05.021
Autor:
Sisi Wu, Xiaoxiao Wang, Mingming Tong, Li Fu, Yanjing Zhang, Jiayi Xu, Xuemei Chen, Hongying Chen, Ruli Li, Yao Wu, Juanjuan Xin, Xin Yan, He Li, Jie Lan, Kunyue Xue, Xue Li, Caili Zhuo, Jiang Wei
Sirt6, a class III NAD+-dependent deacetylase of the sirtuin family, is a highly specific H3 deacetylase and plays important roles in regulating cellular growth and death. The induction of oxidative stress and death are the crucial mechanisms involve
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1f291d7c1dea011b21baecd6f6bdd838
https://doi.org/10.21203/rs.3.rs-263382/v1
https://doi.org/10.21203/rs.3.rs-263382/v1
Autor:
Wei Jiang, Ruli Li, Chaoshu Tang, Juanjuan Xin, Yao Wu, Xue Li, Caili Zhuo, Jinhan He, Hongying Chen, He Li, Kunyue Xue, Xiaoxiao Wang, Sisi Wu, Jie Lan
Publikováno v:
Journal of cellular physiology. 234(10)
Cardiac hypertrophy is the main cause of heart failure and sudden death in patients. But the pathogenesis is unclear. Angiotensin II may contribute to cardiac hypertrophy in response to pressure overload. In angiotensin II-treated cardiomyocytes, the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b38784d3b9390d0e4cf7c1fed10a47c9
https://pubmed.ncbi.nlm.nih.gov/30793300
https://pubmed.ncbi.nlm.nih.gov/30793300
Autor:
Hongying Chen, Juanjuan Xin, Wei Jiang, Xue Li, He Li, Kunyue Xue, Ling Zhu, Sisi Wu, Jie Lan, Ruli Li, Caili Zuo
Publikováno v:
Journal of cellular biochemistry.
Endoplasmic reticulum stress (ERS) is usually involved in tumor development and progression, and anticancer agents have recently been recognized to induce ERS. Cucurbitacin-I showed a potent anticancer action by inducing apoptosis through the inhibit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::aac47954b3ff147c5aea3ae448a43cda
https://pubmed.ncbi.nlm.nih.gov/30277611
https://pubmed.ncbi.nlm.nih.gov/30277611
Autor:
Jie Lan, Caili Zhuo, He Li, Yao Wu, Ruli Li, Xiao-Xiao Wang, Chaoshu Tang, Yu-Yan Cai, Jinhan He, Wang Wang, Wei Jiang, Sisi Wu, Xue Li, Heng-Yu Zhang, Juanjuan Xin, Hongying Chen, Kunyue Xue
Publikováno v:
Journal of molecular and cellular cardiology. 121
In hypertrophic hearts, autophagic flux insufficiency is recognized as a key pathology leading to maladaptive cardiac remodeling and heart failure. This study aimed to illuminate the cardioprotective role and mechanisms of a new myokine and adipokine
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1ad0ced1f81db712d1c442b010bf9c5e
https://pubmed.ncbi.nlm.nih.gov/30053525
https://pubmed.ncbi.nlm.nih.gov/30053525