Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Kun-Ru Zhang"'
Publikováno v:
Kidney & Blood Pressure Research, Vol 46, Iss 2, Pp 219-228 (2021)
Background: Hypertensive nephropathy is one of the major causes of ESRD. Exercise has been considered a nonpathological therapy for hypertension and its complications, yet mechanisms remain unclear. We sought to investigate whether periodic swimming
Externí odkaz:
https://doaj.org/article/639c75e546684fff8e9193db0c4968bf
Publikováno v:
Kidney & Blood Pressure Research, Vol 46, Iss 2, Pp 219-228 (2021)
Background: Hypertensive nephropathy is one of the major causes of ESRD. Exercise has been considered a nonpathological therapy for hypertension and its complications, yet mechanisms remain unclear. We sought to investigate whether periodic swimming
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2c74b0e47de7ba6339f30501cc2451bd
https://www.karger.com/Article/FullText/514680
https://www.karger.com/Article/FullText/514680
Autor:
Zhuan Zhou, Heping Cheng, Jun Ren, Hai-Chang Wang, Feng Gao, Quan-Jiang Zhang, Wenyi Guo, Kun-Ru Zhang, Haifeng Zhang, Qiu-Xia Li
Publikováno v:
Cardiovascular Research. 75:369-380
Objectives Physical activity has been well known to benefit heart function. The improved autonomic nervous activity is considered to be mainly responsible for this beneficial effect. However, the precise mechanism behind the intrinsic myocardial resp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::42f990076f6e6684bb850c7bae498b4a
https://doi.org/10.1016/j.cardiores.2007.04.015
https://doi.org/10.1016/j.cardiores.2007.04.015
Autor:
Feng Gao, Theodore A. Christopher, Wen-Li Yan, Kun-Ru Zhang, Xin L. Ma, Hui Su, Wenyi Guo, Haifeng Zhang, Bernard L. Lopez, Qiu-Xia Li, Hai-Chang Wang
Publikováno v:
Apoptosis. 11:1453-1460
Our previous study has shown that slow or "controlled" reperfusion for the ischemic heart reduces cardiomyocyte injury and myocardial infarction, while the mechanisms involved are largely unclear. In this study, we tested the hypothesis that enhancem
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7148519a4652fa4f1b3a6a4322a478f3
https://doi.org/10.1007/s10495-006-7786-z
https://doi.org/10.1007/s10495-006-7786-z
Autor:
Hai-Tao, Liu, Hai-Feng, Zhang, Rui, Si, Quan-Jiang, Zhang, Kun-Ru, Zhang, Wen-Yi, Guo, Hai-Chang, Wang, Feng, Gao
Publikováno v:
Sheng li xue bao : [Acta physiologica Sinica]. 59(5)
Our previous results have demonstrated that insulin reduces myocardial ischemia/reperfusion (MI/R) injury and increases the postischemic myocardial functions via activating the cellular survival signaling, i.e., phosphatidylinositol 3-kinase (PI3-K)-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::35a83e5c515f0f700d58eee3e3c4fa73
https://pubmed.ncbi.nlm.nih.gov/17940706
https://pubmed.ncbi.nlm.nih.gov/17940706
Autor:
Feng Gao, Quan-Jiang Zhang, Hai-Chang Wang, Qiu-Xia Li, Haifeng Zhang, Hai-Tao Liu, Kun-Ru Zhang, Qiujun Yu, Wenyi Guo
Publikováno v:
Apoptosis : an international journal on programmed cell death. 12(9)
Physical activity has been shown to improve cardiovascular function and to be beneficial to type 2 diabetic patients. However, the effects of aerobic exercise (AE) on myocardial ischemia/reperfusion (MI/R) are largely unclear. Therefore, the aims of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dbb76476193f246a58a19742c4daa479
https://pubmed.ncbi.nlm.nih.gov/17505785
https://pubmed.ncbi.nlm.nih.gov/17505785
Publikováno v:
Journal of Molecular and Cellular Cardiology. 42:S57
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7b1bce1de09c0d63b03f23342df462d7
https://doi.org/10.1016/j.yjmcc.2007.03.767
https://doi.org/10.1016/j.yjmcc.2007.03.767
Autor:
Hai-Chang Wang, Hai-Feng Zhang, Wen-Yi Guo, Hui Su, Kun-Ru Zhang, Qiu-Xia L, Wenli Yan, Ma, Xin L., Lopez, Bernard L., Christopher, Theodore A., Feng Gao
Publikováno v:
Apoptosis; Aug2006, Vol. 11 Issue 8, p1453-1460, 8p, 6 Graphs