Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Kuma Misra"'
Autor:
Zeyuan Ding, Jingxiao Zhao, Raja Devesh Kuma Misra, Fujian Guo, Zhenjia Xie, Xuelin Wang, Xiucheng Li, Jingliang Wang, Chengjia Shang
Publikováno v:
Metals, Vol 13, Iss 11, p 1809 (2023)
A novel YOLOv5 network is presented in this paper to quantify the degree of defects in continuously cast billets. The proposed network addresses the challenges posed by noise or dirty spots and different defect sizes in the images of these billets. T
Externí odkaz:
https://doaj.org/article/0a1de2a4b72149c0a66975889e5bb24b
Autor:
Haitao Jiao, Xinxiang Xie, Xinyi Hu, Longzhi Zhao, Raja Devesh Kuma Misra, Dejia Liu, Yanchuan Tang, Yong Hu
Publikováno v:
Metals, Vol 12, Iss 2, p 354 (2022)
In this study, the effect of the hot-cold rolling process on the evolution of the microstructure, texture and magnetic properties of strip-cast non-oriented electrical steel was investigated by introducing hot rolling with different reductions. The r
Externí odkaz:
https://doaj.org/article/3b19e414503f47ef94263e662202d995
Autor:
Pradeep Kuma Misra
Publikováno v:
Tenth Pan-Commonwealth Forum on Open Learning.
Policy documents in India, most notably National Education Policy 2020, emphasize that the higher education sector may aim for achieving three Sustainable Development Goals (SDGs), particularly tenth (reduced inequalities), fifth (gender equality), a
Autor:
Jonathan A. Fidler, Kuma Misra, Christopher M. Treleaven, Richard L. Sidman, Thomas J. Tamsett, Seng H. Cheng, James Dodge, Tatyana V. Taksir, Channa Bao, Lamya S. Shihabuddin, Michelle Searles
Publikováno v:
Proceedings of the National Academy of Sciences. 110:10812-10817
Metabolic dysfunction is an important modulator of disease course in amyotrophic lateral sclerosis (ALS). We report here that a familial mouse model (transgenic mice over-expressing the G93A mutation of the Cu/Zn superoxide dismutase 1 gene) of ALS e
Autor:
Kuma Misra, Tatyana V. Taksir, Amy M. Richards, Lamya S. Shihabuddin, Bryan Mastis, Seng H. Cheng, Christopher M. Treleaven, Lisa M. Stanek, Sergio Pablo Sardi
Huntington's disease (HD) is a fatal autosomal dominant neurodegenerative disease caused by an increase in the number of polyglutamine residues in the huntingtin (Htt) protein. With the identification of the underlying basis of HD, therapies are bein
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2e7884c10f5cf8063eec756ec1486597
https://europepmc.org/articles/PMC4028091/
https://europepmc.org/articles/PMC4028091/