Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Krzysztof Borecki"'
Publikováno v:
Journal of Medical Science, Vol 85, Iss 3 (2016)
Introduction. In certain pathological states within cardiovascular system, cardiomyocytes may exhibit overexpression of TNF-? that results in induction of myocardial oxidative stress and cardiac cells apoptosis. Thus, they may participate in developm
Externí odkaz:
https://doaj.org/article/5be6663919664bbcb79136f3717c7317
Autor:
Dominika Maciejewska, Igor Łoniewski, Mariusz Kaczmarczyk, Krzysztof Borecki, Ewa Stachowska, Beata Łoniewska, Karolina Skonieczna-Żydecka, Joanna Śliwa-Dominiak, Karolina Kaźmierczak-Siedlecka, Wojciech Marlicz, Katarzyna Janda, Damian Malinowski
Publikováno v:
Journal of Clinical Medicine
Journal of Clinical Medicine, Vol 9, Iss 1788, p 1788 (2020)
Journal of Clinical Medicine, Vol 9, Iss 1788, p 1788 (2020)
We aimed to systematically review the effectiveness of probiotic/synbiotic formulations to counteract cardiometabolic risk (CMR) in healthy people not receiving adjunctive medication. The systematic search (PubMed/MEDLINE/Embase) until 1 August 2019
Autor:
Paulina Zabielska, Karolina Skonieczna-Żydecka, Dominika Jamioł-Milc, Magdalena Sylwia Kamińska, Krzysztof Borecki, Ewa Stachowska, Beata Karakiewicz
Publikováno v:
International Journal of Environmental Research and Public Health, Vol 17, Iss 2, p 400 (2020)
International Journal of Environmental Research and Public Health
International Journal of Environmental Research and Public Health
Iron deficiency have been found to be linked to sleep disorders. Both genetic and environmental factors are risk factors for skewed iron metabolism, thus sleep disruptions in autism spectrum disorders (ASD). The aim of our study was to assess the pre
Autor:
Honorata Mruk-Mazurkiewicz, K. Tejchman, Tomasz Mazur, Joanna Palma, Karina Ryterska, Krzysztof Borecki, Justyna Antoniewicz, Zofia Stachowska, Wojciech Marlicz, Anna Wolska, Przemysław Mijal, Dominika Maciejewska-Markiewicz, Anna Gudan, Natalia Komorniak, Robert Budawski, Maja Czerwińska-Rogowska, Ewa Stachowska, Karolina Skonieczna-Żydecka
Publikováno v:
International Journal of Environmental Research and Public Health
Volume 18
Issue 21
International Journal of Environmental Research and Public Health, Vol 18, Iss 11464, p 11464 (2021)
Volume 18
Issue 21
International Journal of Environmental Research and Public Health, Vol 18, Iss 11464, p 11464 (2021)
Background: Irritable bowel syndrome (IBS) is a chronic functional gastrointestinal disorder (FGID), in which etiology and pathogenesis are not fully explored. There is an ongoing need for more population studies adhering to new ROME IV criteria. In
Autor:
Krzysztof Borecki, Iwona Zawada, Anna Pawinska-Matecka, Nermin Nusret Salkic, Beata Karakiewicz, Grażyna Adler
Publikováno v:
Bosnian Journal of Basic Medical Sciences, Vol 17, Iss 4 (2017)
The role of ABCB1 single nucleotide polymorphisms (SNPs) in the development of Crohn’s disease (CD) remains unclear. Due to inconsistent results of several European population-based studies and limited information on populations from Poland and Bos
Autor:
Krzysztof Borecki, Iwona Zawada, Anna Pawinska-Matecka, Nermin N. Salkic, Beata Karakiewicz, Grażyna Adler
Publikováno v:
Bosnian Journal of Basic Medical Sciences.
The role of ABCB1 single nucleotide polymorphisms (SNPs) in the development of Crohn's disease (CD) remains unclear. Due to inconsistent results of several European population-based studies and limited information on populations from Poland and Bosni
Publikováno v:
International Journal of Environmental Research and Public Health, Vol 16, Iss 9, p 1551 (2019)
International Journal of Environmental Research and Public Health
Volume 16
Issue 9
International Journal of Environmental Research and Public Health
Volume 16
Issue 9
It is suggested that IL-23/IL-17 axis and single nucleotide polymorphisms (SNPs) of IL23R may have crucial role in pathogenesis of Crohn&rsquo
s disease (CD). Thus, we sought to assess the IL23R SNPs contribution to susceptibility and phenotype
s disease (CD). Thus, we sought to assess the IL23R SNPs contribution to susceptibility and phenotype