Zobrazeno 1 - 10
of 66
pro vyhledávání: '"Krysztofinska, Ewelina"'
Autor:
Heuberger, Kathrin, Bailey, Henry J., Burda, Patricie, Chaikuad, Apirat, Krysztofinska, Ewelina, Suormala, Terttu, Bürer, Céline, Lutz, Seraina, Fowler, Brian, Froese, D. Sean, Yue, Wyatt W., Baumgartner, Matthias R.
Publikováno v:
In BBA - Molecular Basis of Disease 1 June 2019 1865(6):1265-1272
Autor:
Mazzon, Michela, Peters, Nicholas E., Loenarz, Christoph, Krysztofinska, Ewelina M., Ember, Stuart W. J., Ferguson, Brian J., Smith, Geoffrey L.
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America, 2013 Jul . 110(30), 12444-12449.
Externí odkaz:
https://www.jstor.org/stable/42712610
Autor:
Simon, Aline C., Simpson, Peter J., Goldstone, Rachael M., Krysztofinska, Ewelina M., Murray, James W., High, Stephen, Isaacson, Rivka L.
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America, 2013 Jan . 110(4), 1327-1332.
Externí odkaz:
https://www.jstor.org/stable/41991772
Autor:
Kakade, Poonam, Ojha, Hina, Raimi, Olawale G., Shaw, Andrew, Waddell, Andrew D., Ault, James R., Burel, Sophie, Brockmann, Kathrin, Kumar, Atul, Ahangar, Mohd Syed, Krysztofinska, Ewelina M., Macartney, Thomas, Bayliss, Richard, Fitzgerald, Julia C., Muqit, Miratul M. K.
Autosomal recessive mutations in the PINK1 gene are causal for Parkinson's disease (PD). PINK1 encodes a mitochondrial localized protein kinase that is a master-regulator of mitochondrial quality control pathways. Structural studies to date have elab
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::012d689d8dbbee8aff6e37244b6bf6fb
Autor:
Kakade Poonam, Ojha Hina, Raimi Olawale G., Shaw Andrew, Waddell Andrew D., Ault James R, Burel Sophie, Brockmann Kathrin, Kumar Atul, Ahangar Mohd Syed, Krysztofinska Ewelina M., Macartney Thomas, Bayliss Richard, Fitzgerald Julia C., Muqit Miratul M. K.
Human autosomal recessive mutations in the PINK1 gene are causal for Parkinson’s disease (PD). PINK1 encodes a mitochondrial localised protein kinase that is a master-regulator of mitochondrial quality control pathways. Structural studies to date h
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::64e221137ab8d4ecc94f4aca9311f845
Akademický článek
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Autor:
Martínez-Lumbreras, Santiago1, Krysztofinska, Ewelina M.1, Thapaliya, Arjun1, Spilotros, Alessandro2, Matak-Vinkovic, Dijana3, Salvadori, Enrico4,5, Roboti, Peristera6, Nyathi, Yvonne6,7, Muench, Janina H.1, Roessler, Maxie M.4, Svergun, Dmitri I.2, High, Stephen6, Isaacson, Rivka L.1 rivka.isaacson@kcl.ac.uk
Publikováno v:
BMC Biology. 7/11/2018, Vol. 16 Issue 1, p1-18. 18p.
Autor:
Krysztofinska, Ewelina Maria
Eukaryotic cells rely on quality control mechanisms to sustain protein homeostasis by regulating protein folding, targeting and degradation. These mechanisms involve the recognition of exposed hydrophobic regions of membrane proteins that have misloc
Externí odkaz:
https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.733450
Autor:
Martínez-Lumbreras, Santiago, Krysztofinska, Ewelina, Thapaliya, Arjun, Spilotros, Alessandro, Matak-Vinkovic, Dijana, Salvadori, Enrico, Roboti, Peristera, Nyathi, Yvonne, Muench, Janina, Roessler, Maxie, Svergun, Dmitri, High, Stephen, Isaacson, Rivka
Figure S7. (A) Size-exclusion chromatography of some different variants of SGTA. Note the unexpected elution volume of the CT construct (red). (B) Dynamic light scattering intensity distributions for CT and CTΔQ constructs showing the size of the mo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ce1a59d8c5927d437ab5c206cb457aa5
Autor:
MartĂNez-Lumbreras, Santiago, Krysztofinska, Ewelina, Thapaliya, Arjun, Spilotros, Alessandro, Matak-Vinkovic, Dijana, Salvadori, Enrico, Roboti, Peristera, Nyathi, Yvonne, Muench, Janina, Roessler, Maxie, Svergun, Dmitri, High, Stephen, Isaacson, Rivka
Figure S2. The SGTA-3xNNP/AAA-V5 mutant stimulates the accumulation of OP91 in discrete cytosolic inclusions. HeLa cells stably expressing OP91 under an inducible promoter were transiently transfected with either a control plasmid (PEX19-V5) or plasm
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::655af2c0fc7d32cfd47faa0ce781fba0