Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Krupen Patel"'
Autor:
Pauline Sebby Ogolla, Jose-Andres C Portillo, Christine L White, Krupen Patel, Bruce Lamb, Ganes C Sen, Carlos S Subauste
Publikováno v:
PLoS Pathogens, Vol 9, Iss 8, p e1003557 (2013)
PKR is well characterized for its function in antiviral immunity. Using Toxoplasma gondii, we examined if PKR promotes resistance to disease caused by a non-viral pathogen. PKR(-/-) mice infected with T. gondii exhibited higher parasite load and wors
Externí odkaz:
https://doaj.org/article/f16ca556eb244d0d8736baae47a32097
Autor:
Jeffrey Tomalka, Sandhya Ganesan, Elaheh Azodi, Krupen Patel, Parth Majmudar, Brian A Hall, Katherine A Fitzgerald, Amy G Hise
Publikováno v:
PLoS Pathogens, Vol 7, Iss 12, p e1002379 (2011)
Candida sp. are opportunistic fungal pathogens that colonize the skin and oral cavity and, when overgrown under permissive conditions, cause inflammation and disease. Previously, we identified a central role for the NLRP3 inflammasome in regulating I
Externí odkaz:
https://doaj.org/article/371a3b5e0d97416090db01053c77df12
Autor:
Cisley Cardwell, Brian A. Hall, Samantha O’Neill, Jeffrey Tomalka, Amy G. Hise, Hema P. Narra, James M. Wilson, Krupen Patel, Elaheh Azodi
Publikováno v:
The Journal of Immunology. 194:1788-1795
Candida is an opportunistic fungal pathogen that colonizes the mucosal tract of humans. Pathogenic infection occurs in the presence of conditions causing perturbations to the commensal microbiota or host immunity. Early innate immune responses by the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::20806da66085bc3d07057b8963c5f3c8
https://doi.org/10.4049/jimmunol.1203239
https://doi.org/10.4049/jimmunol.1203239
Autor:
Cheryl Eitman, L. Ponsky, Ravi Kumar Kyasaram, Leonidas Nikolaos Diamantopoulos, Scott Dawsey, Krupen Patel, Christopher J. Hoimes, Ali Raza Khaki, Ariel Ann Nelson, Prateek Mendiratta, Matthew David Wright, Behtash Ghazi Nezami, Gregory T. MacLennan, Petros Grivas
Publikováno v:
Journal of Clinical Oncology. 37:e16016-e16016
e16016 Background: Prior analyses demonstrated worse outcomes with the BP mUC clinical phenotype. UC molecular subtypes that may correlate with the BP phenotype have been defined, however molecular subtyping is not a readily available standard practi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d9a7e0246b482dcd7b7d840667e7ffa9
https://doi.org/10.1200/jco.2019.37.15_suppl.e16016
https://doi.org/10.1200/jco.2019.37.15_suppl.e16016
Autor:
Christopher J. Hoimes, Ravi Kumar Kyasaram, Matthew M. Cooney, Krupen Patel, L. Ponsky, Behtash Ghazi Nezami, Aman Opneja, Petros Grivas, Pingfu Fu, Gregory T. MacLennan, Patrick Mergler, Pedro C. Barata, Jorge A. Garcia, Matthew David Wright, Ariel Ann Nelson, Cheryl Eitman
Publikováno v:
Journal of Clinical Oncology. 36:e16523-e16523
e16523Background: The clinical phenotype of BP mUC includes patients (pts) with at least as many bone lesions as non-bone lesions. UC molecular subtypes that may correlate with BP phenotype and pro...
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6beb08fcea563876a10af22e58290efd
https://doi.org/10.1200/jco.2018.36.15_suppl.e16523
https://doi.org/10.1200/jco.2018.36.15_suppl.e16523
Publikováno v:
Journal of Bone and Mineral Research. 19:1339-1348
The coordination of cell cycle progression and osteoclast differentiation by RANKL signaling was studied. Experiments with mouse genetic models revealed that RANKL promoted cell cycle withdrawal of osteoclast precursors dependent on the cyclin kinase
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::009e23d6b09f502adb4247ed014aaf00
https://doi.org/10.1359/jbmr.040321
https://doi.org/10.1359/jbmr.040321
Autor:
Amy G. Hise, Stefan A. Schattgen, Katherine A. Fitzgerald, Sivapriya Kailasan Vanaja, Zachary Traylor, Megan E. Ermler, Krupen Patel
Inflammasome activation is gaining recognition as an important mechanism for protection during viral infection. Here, we investigate whether Rift Valley fever virus, a negative-strand RNA virus, can induce inflammasome responses and IL-1β processing
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f7ea27f7cf9c63b8a90b542bc7929e7
https://europepmc.org/articles/PMC3951897/
https://europepmc.org/articles/PMC3951897/
Autor:
Bruce T. Lamb, Jose Andres C Portillo, Ganes C. Sen, Christine L. White, Carlos S. Subauste, Pauline Sebby Ogolla, Krupen Patel
Publikováno v:
PLoS Pathogens, Vol 9, Iss 8, p e1003557 (2013)
PLoS Pathogens
PLoS Pathogens
PKR is well characterized for its function in antiviral immunity. Using Toxoplasma gondii, we examined if PKR promotes resistance to disease caused by a non-viral pathogen. PKR−/− mice infected with T. gondii exhibited higher parasite load and wo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dbb62f52bfbab409c569b4578d7853f1
https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/23990781/?tool=EBI
https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/23990781/?tool=EBI
Autor:
Brian A. Hall, Jeffrey Tomalka, Parth R. Majmudar, Amy G. Hise, Elaheh Azodi, Krupen Patel, Katherine A. Fitzgerald, Sandhya Ganesan
Publikováno v:
PLoS Pathogens, Vol 7, Iss 12, p e1002379 (2011)
PLoS Pathogens
PLoS Pathogens
Candida sp. are opportunistic fungal pathogens that colonize the skin and oral cavity and, when overgrown under permissive conditions, cause inflammation and disease. Previously, we identified a central role for the NLRP3 inflammasome in regulating I
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fb2c85a3a7dcb95841ac774d2ddd6e78
http://europepmc.org/articles/PMC3234225?pdf=render
http://europepmc.org/articles/PMC3234225?pdf=render
Autor:
Sudarshana M. Sharma, Saïd Sif, Michael C. Ostrowski, Kim C. Mansky, Rong Hu, Krupen Patel, Agnieszka Bronisz
Publikováno v:
The Journal of biological chemistry. 282(21)
Transcription factors NFATc1, PU.1, and MITF collaborate to regulate specific genes in response to colony-stimulating factor-1 (CSF-1) and receptor activator of NF-kappaB ligand (RANKL) signaling during osteoclast differentiation. However, molecular
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d9dfe542d7af2cea22184a3c899fb9e5
https://pubmed.ncbi.nlm.nih.gov/17403683
https://pubmed.ncbi.nlm.nih.gov/17403683