Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Krizia-Ivana Udquim"'
Autor:
Ismail D. Legason, Ruth M. Pfeiffer, Krizia-Ivana Udquim, Andrew W. Bergen, Mateus H. Gouveia, Samuel Kirimunda, Isaac Otim, Eric Karlins, Patrick Kerchan, Hadijah Nabalende, Ariunaa Bayanjargal, Benjamin Emmanuel, Paul Kagwa, Ambrose O. Talisuna, Kishor Bhatia, Meredith Yeager, Robert J. Biggar, Leona W. Ayers, Steven J. Reynolds, James J. Goedert, Martin D. Ogwang, Joseph F. Fraumeni, Jr, Ludmila Prokunina-Olsson, Sam M. Mbulaiteye
Publikováno v:
EBioMedicine, Vol 25, Iss C, Pp 58-65 (2017)
Background: Plasmodium falciparum (Pf) malaria infection is suspected to cause endemic Burkitt Lymphoma (eBL), but the evidence remains unsettled. An inverse relationship between sickle cell trait (SCT) and eBL, which supports that between malaria an
Externí odkaz:
https://doaj.org/article/2d035808b01e411893158394fd9cb97c
Autor:
Clara Zettelmeyer, Krizia-Ivana Udquim, Seraph Han-Yin Lin, A. Rouf Banday, Ludmila Prokunina-Olsson
Publikováno v:
Carcinogenesis
Increased exposure to estrogen is associated with an elevated risk of breast cancer. Considering estrogen as a possible mutagen, we hypothesized that exposure to estrogen alone or in combination with the DNA-damaging chemotherapy drug, cisplatin, cou
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fbf0d4aa2942f66e034f22e843bea90e
https://doi.org/10.1093/carcin/bgaa002
https://doi.org/10.1093/carcin/bgaa002
Autor:
Ambrose Talisuna, Hadijah Nabalende, Ismail D. Legason, Kishor Bhatia, Krizia Ivana Udquim, Joseph F. Fraumeni, Andrew W. Bergen, Ariunaa Bayanjargal, Martin D. Ogwang, Steven J. Reynolds, Eric Karlins, Isaac Otim, Benjamin Emmanuel, Robert J. Biggar, Mateus H. Gouveia, Samuel Kirimunda, Ruth M. Pfeiffer, Ludmila Prokunina-Olsson, Leona W. Ayers, Sam M. Mbulaiteye, James J. Goedert, Paul Kagwa, Meredith Yeager, Patrick Kerchan
Publikováno v:
EBioMedicine, Vol 25, Iss C, Pp 58-65 (2017)
EBioMedicine
EBioMedicine
Background Plasmodium falciparum (Pf) malaria infection is suspected to cause endemic Burkitt Lymphoma (eBL), but the evidence remains unsettled. An inverse relationship between sickle cell trait (SCT) and eBL, which supports that between malaria and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53b846ffcfb752d4444b690b13c33a3d
http://www.sciencedirect.com/science/article/pii/S2352396417303912
http://www.sciencedirect.com/science/article/pii/S2352396417303912
Autor:
Taro Shuin, Debra T. Silverman, Bin Zhu, Olusegun O. Onabajo, Candace D. Middlebrooks, Stephen J. Chanock, Montserrat Garcia-Closas, Stella Koutros, Núria Malats, Michiaki Kubo, A. Rouf Banday, Nathaniel Rothman, Jonine D. Figueroa, Krizia Ivana Udquim, Ashley Paquin, Manolis Kogevinas, Neal D. Freedman, Konichi Matsuda, Ludmila Prokunina-Olsson
Publikováno v:
Middlebrooks, C D, Banday, A R, Matsuda, K, Udquim, K-I, Onabajo, O O, Paquin, A, Figueroa, J D, Zhu, B, Koutros, S, Kubo, M, Shuin, T, Freedman, N D, Kogevinas, M, Malats, N, Chanock, S J, Garcia-Closas, M, Silverman, D T, Rothman, N & Prokunina-Olsson, L 2016, ' Association of germline variants in the APOBEC3 region with cancer risk and enrichment with APOBEC-signature mutations in tumors ', Nature Genetics, vol. 48, no. 11, pp. 1330–1338 . https://doi.org/10.1038/ng.3670
High rates of APOBEC-signature mutations are found in many tumors, but factors affecting this mutation pattern are not well understood. Here we explored the contribution of two common germline variants in the APOBEC3 region. SNP rs1014971 was associa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8cd1ab306f0ff649b5bc4feeb358432b
https://hdl.handle.net/20.500.11820/fa78b05e-e2f6-4109-8bc3-a0b3249c3aba
https://hdl.handle.net/20.500.11820/fa78b05e-e2f6-4109-8bc3-a0b3249c3aba
Autor:
A. Rouf Banday, Ariunaa Bayanjargal, Olusegun O. Onabajo, Krizia-Ivana Udquim, Ludmila Prokunina-Olsson
Publikováno v:
Cancer Research. 77:1292-1292
Background: Activity of APOBEC3 enzymes has been identified as a main source of somatic mutations of a specific type (C to T or G substitutions in the TCA or TCT motifs). This mutation pattern, known as the APOBEC signature, has been described in 16
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2a3a772ea306ed42561cc79c9100401b
https://doi.org/10.1158/1538-7445.am2017-1292
https://doi.org/10.1158/1538-7445.am2017-1292
Autor:
A. Rouf Banday, Ludmila Prokunina-Olsson, Krizia-Ivana Udquim, Olusegun O. Onabajo, Adeola Obajemu
Publikováno v:
Cancer Research. 77:598-598
Introduction: Cytidine deaminase activity of APOBEC3 enzymes generates mutations that restrict viral infection and eliminate tumor cells but also contribute to viral and tumor evolution. For example, human immunodeficiency virus (HIV) is hypermutated
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::62773455f8670b924e7dfee13ddfc240
https://doi.org/10.1158/1538-7445.am2017-598
https://doi.org/10.1158/1538-7445.am2017-598