Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Kritton Shay‐Winkler"'
Autor:
Marianne E Emmert, Parul Aggarwal, Kritton Shay-Winkler, Se-Jin Lee, Qingnian Goh, Roger Cornwall
Publikováno v:
eLife, Vol 11 (2022)
Neonatal brachial plexus injury (NBPI) causes disabling and incurable muscle contractures that result from impaired longitudinal growth of denervated muscles. This deficit in muscle growth is driven by increased proteasome-mediated protein degradatio
Externí odkaz:
https://doaj.org/article/b389100cb389497bb75b7e3c45bcc659
Autor:
Qinghang Meng, Bidur Bhandary, Hanna Osinska, Jeanne James, Na Xu, Kritton Shay‐Winkler, James Gulick, Monte S. Willis, Cynthia Lander, Jeffrey Robbins
Publikováno v:
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, Vol 6, Iss 9 (2017)
BackgroundCardiac stress can trigger production of a 40‐kDa peptide fragment derived from the amino terminus of the cardiac myosin‐binding protein C. Cardiac stress, as well as cMyBP‐C mutations, can trigger production of 1 such truncated prote
Externí odkaz:
https://doaj.org/article/dbc27fe9e550438e858ae6824cb461f1
Publikováno v:
Journal of the Pediatric Orthopaedic Society of North America. 4:1-4
Purpose: Neonatal brachial plexus injury (NBPI) causes disabling and incurable muscle contractures, arising from impaired longitudinal growth of denervated muscles. This growth impairment is caused by dysregulation of muscle proteostasis characterize
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e691afe2b38071a3c5b937b5024c31c9
https://doi.org/10.55275/jposna-2022-0046
https://doi.org/10.55275/jposna-2022-0046
Publikováno v:
Febs Letters
Neonatal brachial plexus injury (NBPI) causes disabling and incurable muscle contractures that are driven by impaired growth of denervated muscles. A rare form of NBPI, which maintains afferent muscle innervation despite motor denervation, does not c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6f9b3d4acca2760f6868a7c94d372297
http://europepmc.org/articles/PMC7940581
http://europepmc.org/articles/PMC7940581
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental Biology
Neonatal brachial plexus injury (NBPI) causes disabling and incurable contractures, or limb stiffness, which result from proteasome-mediated protein degradation impairing the longitudinal growth of neonatally denervated muscles. We recently showed in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6f622ecd8f406e1d08a84f8b2c7d4d05
https://pubmed.ncbi.nlm.nih.gov/33236396
https://pubmed.ncbi.nlm.nih.gov/33236396
Autor:
Jeffrey Robbins, Katherine E. Yutzey, James Gulick, Yang Yu, Na Xu, Hanna Osinska, Kritton Shay-Winkler, Patrick M. McLendon
Publikováno v:
Circ Res
Rationale: Compromised protein quality control can result in proteotoxic intracellular protein aggregates in the heart, leading to cardiac disease and heart failure. Defining the participants and understanding the underlying mechanisms of cardiac pro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::15b13ca5c6250dbe212efa03ceb8ffea
https://pubmed.ncbi.nlm.nih.gov/32081062
https://pubmed.ncbi.nlm.nih.gov/32081062
Publikováno v:
SSRN Electronic Journal.
Background: Neonatal brachial plexus injury (NBPI) causes disabling and incurable contractures, or limb stiffness, which result from proteasome-mediated protein degradation impairing the longitudinal growth of neonatally denervated muscles. We recent
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bdb752acb47362462c8cfc53cc3b40f0
https://doi.org/10.2139/ssrn.3684390
https://doi.org/10.2139/ssrn.3684390
Autor:
Keri Anne Drake, Prasad Devarajan, Brian J. Siroky, Kritton Shay-Winkler, Priyanka Parameswaran, LaTawnya Pleasant, Qing Ma, Jeffrey Robbins, Mahima Devarajan
Publikováno v:
American Journal of Physiology-Renal Physiology. 313:F699-F705
The early events that signal renal dysfunction in presymptomatic heart failure are unclear. We tested the hypothesis that functional and mechanistic changes occur in the kidney that precede the development of symptomatic heart failure. We employed a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::71a446478b2fa6ef0d2dd83e6739f027
https://doi.org/10.1152/ajprenal.00505.2016
https://doi.org/10.1152/ajprenal.00505.2016
Autor:
Shenuarin Bhuiyan, Iñigo Valiente-Alandi, Jeffrey Robbins, Burns C. Blaxall, Jeffery D. Molkentin, James Gulick, Qinghang Meng, Bidur Bhandary, Hanna Osinska, Kritton Shay-Winkler, Jeanne James
Publikováno v:
Circulation research. 123(12)
Rationale: Hypertrophic cardiomyopathy occurs with a frequency of about 1 in 500 people. Approximately 30% of those affected carry mutations within the gene encoding cMyBP-C (cardiac myosin binding protein C). Cardiac stress, as well as cMyBP-C mutat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::04110858149736a87479fd09baf31805
https://pubmed.ncbi.nlm.nih.gov/30566054
https://pubmed.ncbi.nlm.nih.gov/30566054