Zobrazeno 1 - 10
of 44
pro vyhledávání: '"Kristina Lejon"'
Publikováno v:
Autoimmunity, Vol 55, Iss 8, Pp 520-528 (2022)
B cells have been shown to be essential for Type 1 diabetes development in the non-obese diabetic mouse, where their contribution as antigen presenting cells has been emphasised. Other important functions for B cells include surface capture of immuno
Externí odkaz:
https://doaj.org/article/203f667192ad4273a7585258532dd8f1
Autor:
Mikael Brink, Anders Lundquist, Andrey Alexeyenko, Kristina Lejon, Solbritt Rantapää-Dahlqvist
Publikováno v:
Arthritis Research & Therapy, Vol 21, Iss 1, Pp 1-14 (2019)
Abstract Background Antibodies and upregulated cytokines and chemokines predate the onset of rheumatoid arthritis (RA) symptoms. We aimed to identify the pathways related to the early processes leading to RA development, as well as potential novel bi
Externí odkaz:
https://doaj.org/article/afbb40af6f3248c498ff7edcf25504f1
Publikováno v:
Autoimmunity, Vol 50, Iss 6, Pp 363-369 (2017)
The underlying cellular and molecular mechanism for the development of Type 1 diabetes is still to be fully revealed. We have previously demonstrated that the NOD mouse, a model for Type 1 diabetes, display a prolonged and enhanced immune response to
Externí odkaz:
https://doaj.org/article/d272106a59924511b83ac030a2b0d8ea
Autor:
Shrikant Shantilal Kolan, Kristina Lejon, Cecilia Koskinen Holm, Rima Sulniute, Pernilla Lundberg, Takashi Matozaki, Per-Arne Oldenborg
Publikováno v:
PLoS ONE, Vol 10, Iss 7, p e0134113 (2015)
B lymphocyte development occurs in the bone marrow, while final differentiation and maturation can occur in both the bone marrow and the spleen. Here we provide evidence that signal regulatory protein α (SIRPα), an Ig-superfamily ITIM-receptor expr
Externí odkaz:
https://doaj.org/article/65755b5100114a648db94e192ee13187
Publikováno v:
PLoS ONE, Vol 7, Iss 4, p e35445 (2012)
Altered metabolism proceeding seroconversion in children progressing to Type 1 diabetes has previously been demonstrated. We tested the hypothesis that non-obese diabetic (NOD) mice show a similarly altered metabolic profile compared to C57BL/6 mice.
Externí odkaz:
https://doaj.org/article/32720852024041d8997e596e9ae42772
Autor:
Mikael Brink, Ewa Berglin, Aladdin J. Mohammad, Anders Lundquist, Inger Gjertsson, Andrey Alexeyenko, Kristina Lejon, Solbritt Rantapää‐Dahlqvist
Funder: Reumatikerförbundet; Id: http://dx.doi.org/10.13039/501100007949
Funder: Stiftelsen Konung Gustaf V:s 80‐årsfond
Funder: Umeå Universitet; Id: http://dx.doi.org/10.13039/501100004885
OBJECTIVE: Antineutrophil cytoplasmic a
Funder: Stiftelsen Konung Gustaf V:s 80‐årsfond
Funder: Umeå Universitet; Id: http://dx.doi.org/10.13039/501100004885
OBJECTIVE: Antineutrophil cytoplasmic a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::43597c5c6bd4663fcb968fe81dda8c4b
http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-211991
http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-211991
Publikováno v:
Scandinavian Journal of Immunology. 97
Autor:
Mikael, Brink, Ewa, Berglin, Aladdin J, Mohammad, Anders, Lundquist, Inger, Gjertsson, Andrey, Alexeyenko, Kristina, Lejon, Solbritt, Rantapää-Dahlqvist
Publikováno v:
Arthritisrheumatology (Hoboken, N.J.).
Antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis (AAV) is a chronic relapsing condition with unknown etiology. To gain insight into molecular processes underlying the disease, we examined biomarkers in blood samples collected prior to
Autor:
Mats Geijer, Torsten Witte, Urban Hellman, Kristina Lejon, Xenofon Baraliakos, Helena Forsblad-d'Elia, Lan Do, Gabriel Granåsen
Publikováno v:
Rheumatology (Oxford, England)
Objectives Antibodies against anti-CD74 are related to axial spondyloarthritis (axSpA). The objectives were (i) to study IgA anti-CD74 in radiographic (r)-axSpA patients in the Backbone cohort and to calculate the sensitivity and specificity of anti-
Publikováno v:
Clinical Rheumatology
Objective Recent studies have demonstrated an altered expression of certain microRNAs in patients with rheumatoid arthritis (RA) as well as their first-degree relatives (FDRs) compared to healthy controls (HCs), suggesting a role of microRNA in the p